difteri bing
TRANSCRIPT
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KAJIAN TEORI
2.1 Definisi Difteri
• Difteria adalah suatu penyakit infeksi mendadak yang disebabkan oleh kuman
Corynebacterium diphteriae. Mudah menular dan yang diserang terutama traktusrespiratorius bagian atas dan ditandai dengan terbentuknya pseudomembran dan
dilepaskannya eksotoksin yang dapat menimbulkan gejala umum dan lokal. (Ilmu
Kesehatan Anak FK UI: 2!"
• Difteri adalah suatu infeksi akut yang disebabkan oleh bakteri penghasil toksik
(racun) Corynebacterium diphteriae. (2#".
• Difteri adalah infeksi saluran pernafasan yang disebabkan oleh Corynebacterium
diphteriae dengan bentuk basil batang gram positif (Jauhari$nuru%in. 2#".
2.2 Klasifikasi
Berdasarkan tingkat keparahannya, Staff lmu !esehatan "nak #!$ membagi penyakit ini
menjadi % tingkat yaitu &
'. nfeksi ringan & bila pseudomembran hanya terdapat pada mukosa hidung dengan
gejala hanya nyeri menelan.
. nfeksi sedang & bila pseudomembran telah menyaring sampai faring (dinding
belakang rongga mulut), sampai menimbulkan pembengkakan pada laring.
%. nfeksi berat & bila terjadi sumbatan nafas yang berat disertai dengan gejala
komplikasi seperti miokarditis (radang otot jantung), paralysis (kelemahan anggota
gerak) dan nefritis (radang ginjal).
Berdasarkan letaknya, digolongkan sebagai berikut &
• Difteri hidung
ejala paling ringan dan paling jarang (*). Mula+mula tampak pilek, kemudian secret yang
keluar tercampur darah sedikit yang berasal dari pseudomembran. enyebaran
pseudomembran dapat mencapai faring dan laring.
• Difteri faring dan tonsil ( Difteri #ausial ).
Difteri jenis ini merupakan difteri paling berat karena bisa mengancam nya-a penderita
akibat gagal nafas. aling sering dijumpai ( /*). ejala mungkin ringan tanpa pembentukan
pseudomembran. Dapat sembuh sendiri dan memberikan imunitas pada penderita.ada
kondisi yang lebih berat dia-ali dengan radang tenggorokan dengan peningkatan suhu tubuh
yang tidak terlalu tinggi, pseudomembran a-alnya hanya berupa bercak putih keabu+abuan
yang cepat meluas ke nasofaring atau ke laring, nafas berbau, dan ada pembengkakan
regional leher tampak seperti leher sapi (bull’s neck). Dapat terjadi sakit menelan, dan suara
serak serta stridor inspirasi -alaupun belum terjadi sumbatan laring.
• Difteri laring dan trakea
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0ebih sering merupakan penjalaran difteri faring dan tonsil, daripada yang primer. ejala
gangguan nafas berupa suara serak dan stridor inspirasi jelas dan bila lebih berat timbul sesak
nafas hebat, sianosis, dan tampak retraksi suprasternal serta epigastrium. "da bull1s neck,
laring tampak kemerahan dan sembab, banyak sekret, dan permukaan ditutupi oleh
pseudomembran. Bila anak terlihat sesak dan payah sekali perlu dilakukan trakeostomi
sebagai pertolongan pertama.
• Difteri kutaneus dan 2aginal
Dengan gejala berupa luka mirip saria-an pada kulit dan 2agina dengan pembentukan
membrane diatasnya. 3amun tidak seperti saria-an yang sangat nyeri, pada difteri, luka yang
terjadi justru tidak terasa apa+apa. Difteri dapat pula timbul pada daerah konjungti2a dan
umbilikus.
• Diphtheria !ulit, !onjungti2a, 4elinga
Diphtheria kulit berupa tukak di kulit, tepi jelas dan terdapat membran pada dasarnya.
!elainan cenderung menahun. Diphtheria pada mata dengan lesi pada konjungti2a berupa
kemerahan, edema dan membran pada konjungti2a palpebra. ada telinga berupa otitis
eksterna dengan sekret purulen dan berbau.
2.& Anat'mi %an Fisi'l'i
• Anat'mi )istem *erna*asan
Saluran pernapasan terdiri dari rongga hidung, faring, laring, trakea, broncus dan paru.
(3elson, 5'5)
• Saluran pernafasan bagian atas &
• 6ongga hidung
7idung berfungsi sebagai penyaring kotoran, melembabkan serta menghangatkan udara yang
dihirup ke dalam paru 8 paru
• #aring
#aring adalah struktur yang menghubungkan hidung dengan rongga mulut ke laring.. #ungsi
utamanya adalah untuk menyediakan saluran pada traktus respiratoriun dan digestif.
• 0aring
0aring adalah struktur epitel kartilago yang menghubungkan faring dan trakhea. #ungsi
utamanya adalah untuk memungkinkan terjadinya lokalisasi. 0aring juga melindungi jalan
nafas ba-ah dari obstruksi benda asing dan memudahkan batuk.
) Saluran pernafasan bagian ba-ah &
• 4rakhea
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Disokong oleh cincin tulang ra-an yang berbentuk seperti sepatu kuda yang panjangnya
kurang lebih / inci, tempat dimana trakea bercabang menjadi bronkus utama kiri dan kanan
dikenal sebagai karina.
• Bronkus
4erdiri atas bagian yaitu broncus kanan dan kiri. Bronkus kanan lebih pendek dan lebar,
merupakan kelanjutan dari trakhea yang arahnya hampir 2ertikal. Bronchus kiri lebih panjang
dan lebih sempit, merupakan kelanjutan dari trakhea dengan sudut yang lebih tajam.
• "l2eoli
aru terbentuk oleh sekitar %55 juta al2eoli. 4erdapat tiga jenis sel 8 sel al2eolar, sel al2eolar
tipe adalah sel epitel yang membentuk dinding al2eolar. Sel al2eolar tipe
sel8sel yang aktif secara metabolik, mensekresi surfactan, suatu fosfolipid yang melapisi
permukaan dalam dan mencegah al2eolar agar tidak kolaps. Sel al2eolar tipe adalah
makrofag yang merupakan sel 8 sel fagositosis yang besar yang memakan benda asing dan
bekerja sebagai mekanisme pertahanan penting.
• aru
aru+paru merupakan organ elastic berbentuk kerucut yang terletak dalam rongga torak atau
dada. !edua paru+paru saling terpisah oleh mediasinum central yang mengandung jantung
pembulu+pembulu darah besar.
0etak paru+paru dirongga dada dibungkus oleh selaput yang bernama pleura.
leura dibagi menjadi dua&
'). leura 9isceral (selaput dada pembungkus) yaitu selaput paru yang langsung
membungkus paru+paru:
). leura arietal yaitu selaput yang melapisi rongga dada sebelah luar. "ntara kedua pleura
ini terdapat rongga (ka2um) yang disebut ka2um pleura.
ada keadaan normal, ka2um pleura ini 2akum (hampa udara) sehingga paru+paru dapat
berkembang kempis dan juga terdapat sedikit cairan (eksudat) yang berguna untukmeminyaki permukaannya (pleura), menghindarkan gesekan antara paru+paru dan dinding
dada se-aktu ada gerakan bernafas.
• Fisi'l'i)istem +ernafasan
!etika udara atmosfer mencapai al2eoli, oksigen akan bergerak dari al2eoli melintasi
membran al2eolar kapiler dan menuju sel darah merah. Sistem sirkulasi kemudian akan
memba-a oksigen yang telah berikatan dengan sel darah merah menuju jaringan tubuh,
dimana oksigen akan digunakan sebagai bahan bakar dalam proses metabolisme. ertukaran
oksigen dan karbon dioksida pada membran al2eolar kapiler dikenal dengan istilah difusi
pulmonal. Setelah proses pertukaran gas selesai (kadar karbondioksida yang rendah) akanmenuju sisi kiri jantung, dan akan dipompakan ke seluruh sel dalam tubuh.
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Saat mencapai jaringan, sel darah merah yang teroksigenasi ini akan melepaskan ikatannya
dengan oksigen dan oksigen tersebut digunakan untuk bahan bakar metabolisme. ;uga
karbondioksida akan masuk sel darah merah. Sel darah merah yang rendah oksigen dan tinggi
karbondioksida akan menuju sisi kanan jantung untuk kemudian dipompakan ke paru+paru.
7al yang sangat penting dalam proses ini adalah bah-a al2eoli harus terus menerus
mengalami pengisian dengan udara segar yang mengandung oksigen dalam jumlah yangcukup.
( Sumber & http&
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• Sakit dan pembengkakan pada tenggorokan
• Mual, muntah , sakit kepala.
• "danya pembentukan selaput di tenggorokan ber-arna putih ke abu abuan kotor.
• !aku leher
!eluhan a-al yang paling sering adalah nyeri tenggorokan, nausea, muntah, dan disfagia.
Selain itu ditandai dengan adanya membran semu di tonsil dan di sekitarnya, serta pelepasan
eksotoksin, yang dapat menimbulkan gejala umum (seperti penyakit infeksi) atau local
(seperti tampak keluhan nyeri)
2.3 +at'fisi'l'i
Biasanya bakteri berkembang biak pada atau di sekitar permukaan selaput lendir mulut atau
tenggorokan dan menyebabkan peradangan. Bila bakteri sampai ke hidung, maka hidung
akan berair. eradangan bisa menyebar dari tenggorokan ke pita suara (laring) dan
menyebabkan pembengkakan sehingga saluran udara menyempit dan terjadi gangguan
pernafasan.
Bakteri ini ditularkan melalui percikan ludah dari batuk penderita atau benda maupun
makanan yang telah terkontaminasi oleh bakteri. !etika telah masuk dalam tubuh, bakteri
melepaskan toksin atau racun. 4oksin ini akan menyebar melalui darah dan bisa
menyebabkan kerusakan jaringan di seluruh tubuh, terutama jantung dan saraf.
"kibat yang ditimbulkan oleh penyakit ini banyak bergantung pada efek eksotoksin yang
diproduksi. 4oksin menghambat pembuatan protein sel sehingga sel mati. 3ekrosis jaringan pada tempat menempelnya kuman akan menunjang perkembang+biakan kuman dan produksi
toksin selanjutnya, serta pembentukan membran yang melekat erat pada dasarnya.
Basil hidup dan berkembang biak pada traktus respiratorius bagian atas, terlebih bila terdapat
peradangan kronis pada tonsil, sinus dan lain+lain. 4etapi -alaupun jarang, basil dapat pula
hidup pada daerah 2ul2a, telinga dan kulit.
ada tempat ini basil membentuk pseudomembran dan melepaskan eksotoksin.
seudomembran dapat timbul lokal atau kemudian menyebar dari faring atau tonsil ke laring
dan seluruh traktus respiratorius bagian atas sehingga menimbulkan gejala yang lebih berat.
!elenjar getah bening sekitarnya akan mengalami hiperplasia dan mengandung toksin.
2.! K'm*likasi
!omplikasi yang timbul pada pasien difteri &
'. Miokarditis
• biasanya timbul akhir minggu kedua atau a-al minggu ketiga perjalanan penyakit
• emerikasaan #isik & rama derap, bunyi jantung melemah atau meredup, kadang+
kadang ditemukan tanda+tanda payah jantung.
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. !olaps perifer
%. bstruksi jalan nafas dengan segala akibatnya, bronkopneumonia dan atelektasis
=. $rogenital & dapat terjadi nefritis
enderita difteri ('5*) akan mengalami komplikasi yg mengenai sistem susunan saraf
terutama sistem motorik. 4erjadi pada akhir minggu pertama perjalanan penyakit.
2.# +enatalaksanaan
• engobatan $mum (Buku kuliah ilmu kesehatan anak #!$, 'AAA) &
• "nti Diphteri Serum ("DS) diberikan sebanyak 5.555$
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• ada urine terdapatnya albuminuria ringan
6A6 II
A)U7AN KE+ERA8ATAN TEORITI)
&.1 +enka5ian
&.1.1 I%entitas Klien
Meliputi nama, umur, no M6, pekerjaan, alamat, agama, cara masuk, ri-ayat alergi, tanggal
masuk 6S dan lain+lain.
&.1.2 Ri9aat Kesehatan
'. 6i-ayat !esehatan Dahulu
Biasanya klien mengalami peradangan kronis pada tonsil, sinus, faring, laring, dan saluran
nafas atas dan mengalami pilek dengan sekret bercampur darah yang disebabkan oleh bakteri
Corynebacterium diphteriae.
. 6i-ayat !esehatan Sekarang
• Biasanya klien mengalami demam yang tidak terlalu tinggi, lesu, pucat, sakit kepala,
anoreksia
• Biasanya klien mengeluh sakit menelan, menggigil, malaise, sakit tenggorokan, batuk.
%. 6i-ayat !esehatan !eluarga
Biasanya ada keluarga yang mengalami difteri jadi ada kemungkinan besar anaknya akan
menderita penyakit yang sama.
=. 6i-ayat erinatal dan 3eonatal
E 7amil
$ntuk mengetahui !ondisi ibu selama hamil, periksa kehamilan dimana dan berapa kali,
serta mendapatkan apa saja dari petugas kesehatan selama hamil.
E ersalinan
• $ntuk mengetahui cara persalinan, ditolong oleh siapa, adakah penyulit selama
melahirkan seperti perdarahan.
• !aji dimana klien dilahirkan, berat badan, panjang badan bayi.
E 3eonatal
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$ntuk mengetahui apakah bayi minum "S atau asi, berapa BB 0ahir, B lahir, apakah saat
lahir bayi langsung menangis. 6i-ayat imunisasi anak dan kesehatan keluarga.
"pakah ri-ayat imunisasi pada anak lengkap
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ntegumen& bersih
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efektif b
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8 ain le2el
8 ain control
4ujuan & nyeri berkurang atau
hilang.
!riteria hasil &
o asien dapat mengatakan nyeri
yang dirasakan
o 3yeri berkurang
o Hajah tidak meringis.
o Skala nyeri berkurang.( 5+ )
o 449 normal
"ktifitas !epera-atan &
8 0akukan pengkajian nyeri secara
menyeluruh meliputi lokasi, durasi,
frekuensi, kualitas, keparahan nyari danfactor pencetus nyeri
8 bser2asi ketidaknyamanan non
2erbal
. ain control
"kiti2itas !epera-atan &
8 "jarkan untuk menggunakan teknik
non farmakologi misal relaksasi, guided
imageri, terapi musik dan distraksi
8 !endalikan factor lingkungan yang
dapat mempengaruhi respon pasien
terhadap ketidaknyamanan misal suhu,
lingkungan, cahaya, kegaduhan.
8 !olaborasi& pemberian analgetik
sesuai indikasi
Sumber & ( Buku saku diagnosa dengan inter2ensi 3C dan criteria hasil 3C< ;udith M.
Hilkinson & ahli bahasa, Hidya-ati.. Ket al.L & editor edisi Bahasa ndonesia, ny Meiliya,
Monica ster. 8 d. . 8 ;akarta & C, 55>.)
&., Im*lementasi
N' D; Ke*era9atan Im*lementasi Ke*era9atan
'ola nafas napas tidak efektif
b
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b
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2.1 Definiti'n Di*htheria
• Diphtheria is an infectious disease caused by the bacteria suddenly Corynebacterium
diphteriae. Contagious and attacked mainly the upper respiratory tract and is
characteriIed by the formation and release of pseudomembranous eGotoGin can cause
local and general symptoms. (+e%iatri0s FK UI: 2!"
•
Diphtheria is an acute infection caused by bacteria producing toGic (poison)Corynebacterium diphteriae. (2#".
• Diphtheria is a respiratory infection caused by Corynebacterium diphteriae -ith stem
gram+positi2e bacilli form (Jauhari$ nuru%in. 2#".
2.2 -lassifi0ati'n
Based on the se2erity, Staff ediatrics #aculty of medicine di2ide the disease into three
le2els, namely&
'. Mild infections& -hen pseudomembranous only found in the nasal mucosa -ithsymptoms only painful s-allo-ing.
. nfection -as& -hen pseudomembranous has filtered through the pharynG (the back
-all of the oral ca2ity), to cause s-elling of the larynG.
%. Se2ere infections& in case of blockage by hea2y breathing accompanied by symptoms
of complications such as myocarditis (inflammation of the heart muscle), paralysis
(limb -eakness) and nephritis (kidney inflammation).
Based on its location, are classified as follo-s&
• diphtheria nose
Symptoms lightest and most rarely (*). "t first look cold, then the secret is out miGed -ith
a little blood coming from pseudomembranous. seudomembranous deployment can reach
the pharynG and larynG.
• Diphtheria pharynG and tonsils (diphtheria #ausial).
Diphtheria Diphtheria of this type is the most serious because it could threaten the li2es of
patients due to respiratory failure. 4he most freJuent (/*). Symptoms may be mild -ithout
pseudomembrane formation. Can heal itself and pro2ide immunity to the more se2ereconditions penderita.ada begins -ith a sore throat -ith increased body temperature is not
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too high, pseudomembranous originally just a grayish+-hite spots that Juickly eGpanded into
the nasopharynG or larynG, halitosis, and there regional s-elling of the neck looks like the
neck of a co- (bull's neck). 4here may be pain in s-allo-ing, and hoarseness, and
inspiratory stridor laryngeal obstruction although it has not happened yet.
• Diphtheria of the larynG and trachea
More often the propagation of diphtheria pharynG and tonsils, rather than the primary. Breath
disorder symptoms such as hoarseness and stridor ob2ious inspirations and -hen more se2ere
shortness of breath arise terrific, cyanosis, and appear retraction suprasternal and epigastric.
3o bulls neck, larynG appear red and s-ollen, a lot of secretions, and the surface is co2ered
by pseudomembranous. Hhen children look cro-ded and chi2ied need to do a tracheostomy
as first aid.
• Diphtheria cutaneous and 2aginal
Hith symptoms such as thrush+like sores on the skin and 2agina -ith the formation of
membrane thereon. But unlike canker sores are painful, the diphtheria, -ounds that happened
just do not feel anything. Diphtheria can also occur on areas of the conjuncti2a and the
umbilicus.
• Diphtheria skin, conjuncti2a, ar
Diphtheria form of skin ulcers on the skin, clear edges and there membrane basically. 4he
disorder tends to chronic. Diphtheria in the eye -ith conjuncti2al lesion in the form of
redness, edema and palpebral conjuncti2a membrane on. n the form of otitis eGterna ear -ith
purulent secretions and smell.
2.& Anat'm an% +hsi'l'
• The anat'm 'f the res*irat'r stem
6espiratory tract consists of the nasal ca2ity, pharynG, larynG, trachea, and lungs broncus.
(3elson, 5'5)
• $pper respiratory tract&
• nasal ca2ity
4he nose acts as a filter dirt, moisten and -arm the air that is inhaled into the lungs + lungs
• faring
4he pharynG is the structure that connects the mouth to the nasal ca2ity larynG .. ts main
function is to pro2ide a channel on respiratoriun and digesti2e tract.
• larynG
4he larynG is the epithelial structure of cartilage that connects the pharynG and trachea. ts
main function is to allo- localiIation. 4he larynG also protect the lo-er air-ay obstruction
foreign body and eases cough.
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) lo-er respiratory Channels&
• trachea
Supported by a ring of cartilage that is shaped like a horseshoe that is less than / inches,
-here the trachea branches into a left and right main bronchus kno-n as !arina.
• bronchi
Consisting of t-o parts, namely broncus right and left. 6ight bronchus is shorter and -ider, is
a continuation of the trachea, -hich is directed almost 2ertically. 0eft bronchus is longer and
narro-er, a continuation of the trachea -ith a sharper angle.
• al2eoli
0ung formed by about %55 million al2eoli. 4here are three types of cells + al2eolar cells,
al2eolar type cells are epithelial cells that form the al2eolar -all. 4ype al2eolar cells
cells that are metabolically acti2e, secreting surfactan, a phospholipid that lines the inner
surface and in order not to pre2ent al2eolar collapse. "l2eolar type cells are macrophages,
-hich are cells + the cells that eat phagocytosis large foreign objects and -orks as an
important defense mechanism.
• lung
4he lungs are elastic cone+shaped organ located in the piston ca2ity or chest. Both lungs
separated from each other by a central mediasinum containing heart+pembulu large blood2essels.
4he layout of the lungs chest ca2ity co2ered by a membrane called the pleura.
4he pleura is di2ided into t-o&
'). 9isceral pleura (the lining of the chest -rapper) -hich are immediately -rapped pleura
lungs:
). arietal pleura is the membrane that lines the chest ca2ity outside. 4here are bet-een the
t-o pleural ca2ity (ca2ity) is called pleural ca2ity.
n normal circumstances, the pleural ca2ity is a 2acuum (2acuum) so that the lungs can
de2elop flat and there is also a bit of fluid (eGudates) are useful to lubricate the surface
(pleura), to a2oid friction bet-een the lung and the chest -all -hen there is a mo2ement to
breathe.
• +hsi'l' Res*irat'r )stem
Hhen the atmospheric air reaches the al2eoli, oGygen mo2es from the al2eoli across the
al2eolar capillary membrane and into the red blood cells. 4he circulatory system -ould then
carry the oGygen that has been bound to red blood cells to the tissues of the body, -hereoGygen is used as fuel in the process of metabolism. 4he eGchange of oGygen and carbon
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dioGide in the al2eolar capillary membrane kno-n as pulmonary diffusion. "fter the gas
eGchange process is complete (lo-er carbon dioGide le2els) going to-ards the left side of the
heart, and -ill be pumped to the rest of the cells in the body.
Hhen it reaches the net-ork, oGygenated red blood cells -ill be untied -ith oGygen and the
oGygen used to fuel metabolism. "lso carbon dioGide -ill enter the red blood cells. 6ed blood cells are lo- in oGygen and high in carbon dioGide going to-ards the right side of the
heart and then pumped to the lungs. t is 2ery important in this process is that the al2eoli must
continue to ha2e filling -ith fresh air containing oGygen in sufficient Juantities.
(Source& http&5? C for '5 minutes, lasting se2eral -eeks in ice,
-ater, milk and mucus had dried up. 4here are three types of basil that forms gra2is, mitis,
intermedius and on the basis of differences in the colonies form blood agar cultures
containing potassium telurit.
Diphtheria bacillus has the properties&
• sedomembran perilous formed at sides, bleed easily, and canescent co2ering areas
terkena.terdiri of fibrin, leukocytes, necrotic tissue and bacteria.
• GotoGin issued a highly malignant and can be toGic to the net-ork after a fe- hours
absorbed and pro2ides an o2er2ie- of net-ork changes are typical, especially in the
heart muscle, kidney and ner2e tissue.
2. -lini0al anifestati'ns
• #e2er, increased body temperature to %@.A degrees Celsius,
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• Coughs and colds are mild.
• ain and s-elling in the throat
• 3ausea, 2omiting, headache.
• 4he formation of membranes in the throat is -hite to grayish ash dirty.
• stiff neck
4he most common early complaint is sore throat, nausea, 2omiting, and dysphagia.
"dditionally characteriIed by a pseudo membrane on the tonsils and in the 2icinity, as -ell as
the release of eGotoGin, -hich can cause general symptoms (such as infectious diseases) or
local (as seen complaints of pain)
2.3 +ath'*hsi'l'
$sually the bacteria multiply on or near the surface of the mucous membranes of the mouthor throat and cause inflammation. Hhen bacteria get into the nose, the nose -ill be runny.
4he inflammation can spread from the throat to the 2oice boG (larynG) and cause s-elling
that narro-s air-ays and respiratory disorders occur.
4he bacteria are spread through sali2a splashes of coughing patients or objects or food that
has been contaminated by bacteria. Hhen it has entered the body, the bacteria release toGins
or poisons. 4his toGin -ill spread through the blood and can cause tissue damage throughout
the body, especially the heart and ner2es.
4he impact caused by this disease depend a lot on the effects of eGotoGin produced. 4oGininhibits protein production of cells so that the cells die. 4issue necrosis at the site of
attachment of the bacteria -ill support the proliferation of germs and subseJuent toGin
production, as -ell as the formation of membrane firmly attached to its base.
Basil li2e and breed in the upper respiratory tract, especially -hen there is a chronic
inflammation of the tonsils, sinus and others. But rarely, basil can also li2e on the 2ul2a, ears
and skin.
"t this point basil pseudomembranous form and releases an eGotoGin. seudomembranous
may arise locally or later spread from the pharynG or larynG and tonsil to the entire upper
respiratory tract, causing more se2ere symptoms. Surrounding lymph nodes -ill undergohyperplasia and contain toGins.
2.! -'m*li0ati'ns
Complications arise in patients -ith diphtheria&
'. myocarditis
• usually arises end of the second -eek or the beginning of the third -eek of the course
of disease
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• hysical eGamination& galloping rhythm, heart sounds -eak or dimmed, sometimes
found signs of heart trouble.
. collapsed peripheral
%. "ir-ay obstruction -ith all its conseJuences, bronchopneumonia and atelectasis
=. $rogenital& nephritis can occur
Diphtheria patients ('5*) -ill de2elop complications that the ner2ous system, especially the
motor system. ccurs at the end of the first -eek of the course of disease.
2.# Treatment
• eneral Medicine (Books college of pediatrics faculty of medicine, 'AAA)&
•
"nti Diphteri Serum ("DS) is gi2en 5.555$ < day di2e days in a ro-, -ith the pre2iously tested the skin and eyes.
• "ntibiotics, procaine penicillin /5.555$ < kg < day to % days free of heat. n patients
-ho do trach, added chloramphenicol / mg < kg < day, di2ided four doses.
• Corticosteroids, intended to pre2ent complications of myocarditis is 2ery dangerous.
May be gi2en prednisone mg < kg < day for % -eeks and then discontinued gradually
• "ccording 3gastiyah ('AA)& Management of nursing patients in -hich patients
treated -ith diphtheria dikamar insulation co2ered. ersonnel should -ear a skort
(aprons) and the mask must be replaced e2ery turn of duty or -hen dirty. Should also be a2ailable supplies of hand -ashing, disinfectant soap, -ashcloth or to-el dry. "lso
the place to soak cutlery filled -ith disinfectant.
2.4 Inestiati'ns
'. Schick test
4he skin test is used to determine the immune status of patients. #or this eGamination in use a
dose '
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-7A+TER II
NUR)IN< T7EORETI-A=
&.1 Assessment
&.1.1 -lient I%entit
ncludes the name, age, no M6, occupation, address, religion, logging in, and allergy history,
date of admission and others.
&.1.2 7ealth 7ist'r
'. #ormerly 7ealth 7istory
$sually the client is eGperiencing a chronic inflammation of the tonsils, sinuses, pharynG,
larynG, and upper respiratory tract and colds -ith secretions miGed -ith blood -hich is
caused by the bacterium Corynebacterium diphteriae.
. 7ealth 7istory 3o-
• $sually clients ha2e a fe2er that is not too high, lethargy, pale, headache, anoreGia
• $sually clients complain of pain in s-allo-ing, chills, malaise, sore throat, cough.
%. #amily 7ealth 7istory
$sually there are families -ho ha2e diphtheria so no child -ill likely suffer from the same
disease.
=. erinatal and 3eonatal 7istory
E regnant
4o determine the condition of the mother during pregnancy, pregnancy check -here and ho-
many times, as -ell as get any health -orker during pregnancy.
E Childbirth
• 4o determine the mode of deli2ery, helped by -hom, is there any complications
during childbirth, such as bleeding.
• "ssess -here the client -as born, -eight, body length baby.
E 3eonatal
4o find out if the baby drinks milk or asi, ho- birth -eight, birth B, -hether at birth the
baby started to cry < no.
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/. " history of gro-th and de2elopment
ro-th and de2elopment of motor, sensory clients -ith diphtheria usually interrupted
breathing so hard to s-allo-, accompanied by fe2er, chills, malaise, sore throat, cough.
>. Childs immuniIation history and family health.
s the childs immuniIation history in full < no.
&.1.& +hsi0al E;aminati'n
n 449 obtained&
'. Body temperature F%@.A N C
. 6espiratory& > G < min (increase)
%. Blood pressure& '55
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. palpation:
7ead& palpable lump abnormal < no
3eck& tyorid palpable enlarged glands, glands and enlargement of 2eins lymfe jugolaris < no.
Chest& symmetric < no, looks lump abnormal < no, irregular breath < no.
Stomach& palpable lump abnormal < not ..
ntegumentary& dry < damp, ugly turgor < no
%. auscultation:
Chest& 6onchi and -heeIing sound < no
"bdomen& audible bo-el < no
=. percussion:
atellar refleG left < right positi2e < no
Stomach& 3o bloating < no
&.2 Nursin Dian'sis
#rom some data in dapatka in diphtheria patients, -e conclude that the diagnosis can arise&
'. Breath breathing pattern ineffecti2e b < d edema of the larynG.
. 4he imbalance in nutrition less than body reJuirements b < d anoreGia.
%. "cute pain b < d the inflammatory process.
Source& (3ursing Diagnosis& defiisi and Classification 55A+5'' < editor, 4. 7eather
7erdman: linguists, Made Surma-ati, D-i Hidiatri, stu 4iar: editor of the ndonesian
edition, Monica sther + ;akarta& C 5'5)
Nursin Interenti'ns &.&
Nursin %; Nursin *lan
O/5e0ties An% -riteria Results Interenti'n
'.ola breath breathing is
effecti2e b < d edema of
the larynG.
3C&
+ 6espiratory status& "ir-ay
patency.
+ 9ital sign status
bjecti2e& patient breathing pattern back to normal.
'. 6espiratory status& "ir-ay patency
"cti2ity, 3ursing&
+ bser2ation of signs + 2ital signs.
+ osition the patient semi+#o-ler.
+ nstruct the patient not to mo2e too
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Gpected outcomes&
o 4he freJuency of respiratory dlm
normal range
o 4he rhythm of the breath as
eGpected.
o Spending sputum in the air-ay
o 3o additional breath sounds
o Breathing easy
o 3o dyspnea
much.
+ 4each the patient to cough effecti2ely
+ Collaboration -ith the medical team in
the deli2ery of Gygen therapy
. mbalance nutrition
less than body
reJuirements b < danoreGia.
3C&
+ 3utritional status& "deJuacy of
nutrient
+ 3utritioal status& food and fluid
intake
bjecti2e& 3utrition clients can be
met.
Gpected outcomes&
o Clients can find out about her
illness.
o 4here is an interest and appetite.
o ating as needed
o BB increased.
'. 3utritional status& food and fluid
intake
3ursing "cti2ities&
+ Monitor caloric intake and the Juality
of food consumption.
+ i2e small portions and the food issoft < mushy.
+ i2e the meal according to taste.
+ Heigh BB e2ery day
%. "cute pain b < d theinflammatory process
3C&
+ ain le2el
+ ain control
bjecti2e& pain diminished or
disappeared.
Gpected outcomes&
'. ain le2el
3ursing "cti2ity&
+ erform a thorough assessment of pain
include the location, duration,
freJuency, Juality, se2erity of pain nyari
and trigger factor
+ bser2ation of non+2erbal discomfort
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atients can say the pain felt
o ain is reduced
o face not to grimace.
o Scale pain -as reduced. (5+)
o 449 normal
. ain control
"kiti2itas 3ursing&
+ 4each for using non+pharmacologicaltechniJues eg relaGation, guided
imageri, music therapy and distraction
+ Control of en2ironmental factors that
can influence patient response to
discomfort eg temperature, en2ironment,
light, noise.
+ Collaboration& analgetik according to
indications
Source& (ocket Books diagnosis -ith inter2ention criteria 3C and 3C results < ;udith M.
Hilkinson& linguists, Hidya-ati .. Ket al.L& ndonesian edition editor, ny Meiliya, Monica
sther. + d. . + ;akarta& C, 55>.)
&., Im*lementati'n
N'. Nursin %; im*lementati'n 'f Nursin
'
Breath breathing pattern
ineffecti2e b < d edema of the
larynG.
+ bser2e signs + 2ital signs.
+ ositioning the patient semi+#o-ler.
+ "d2ise the patient not to mo2e too much.
+ 4each the patient to cough effecti2ely
+ Mengkolaborasi -ith another medical team, Gygen
therapy deli2ery
4he imbalance in nutrition
less than body reJuirements
b < d anoreGia.
+ Monitor the caloric intake and the Juality of food
consumption.
+ ro2ide small ser2ings and soft foods < mushy.
+ ro2ides meal according to taste.
+ Considering BB e2ery day
% "cute pain b < d the
inflammatory process
+ Conduct a thorough assessment of pain include the
location, duration, freJuency, Juality, se2erity of pain
nyari and trigger factor
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+ bser2ing non+2erbal discomfort
+ 4each you to use non+pharmacological techniJues eg
relaGation, guided imageri, music therapy and
distraction
+ Controlling en2ironmental factors that can influence
patient response to discomfort eg temperature,
en2ironment, light, noise.
+ Mengkolaborasi& pro2iding analgesic according to
indications
&. Ealuati'n
Do after the implementation, the e2aluation of our patients, namely&
• atient breathing pattern back to normal, and the patient does not eGperience anymore
dypnea
• 3utrition of patients can be met, and the -eight can be increased
• Gperienced pain in patients can be reduced, and also the pain -ill disappear
6I6=IO
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