Download - Materi Kmb i Kardio 2010
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AMI (AKUT MIOCARD INFARK)
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PENGERTIAN Kematian jaringan miokardium yang diakibatkan oleh
kerusakan atau tidak adekuatnya aliran darah koronermiokard. Hal ini karena penyempitan ataupenyumbatan karena atherosklerosis atau penurunanaliran darah karena shock / pendarahan.
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ETIOLOGI Sebagian besar aterosklerosis
Emboli (gelembung udara yang terbawa
dalam aliran darah) Trombus (bekuan darah dalam pembuluh
darah)
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MANIFESTASI KLINISNyeri dada
Takikardi
Kemungkinan perubahan TDBerkurangnya aliran perifer
Disritmia
Diaporesis
Kulit dingin dan lembab
Pada sistem lain (Takipnu, dispnu, napas pendek,
mual,cemas)
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PENATALAKSANAAN MEDIS Terapi trombolitik
Streptokinase
Anisoylated plasminogen streptokinase activator complex
(APSAC) Tissue plasminogen activator (t-PA)
Reteplase
Analgesik
Nitrogliserin Morfin sulfat
Antidisritmik
Medikasi lain
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…….
Beta bloker (propanolol, atenolol, metoprolol)
==> menurunkan heart rate, mengurangi kerja
jantung dan kebutuhan oksigen jantung Antikoagulan : aspirin dan persantine ==>
menurunkan formasi bekuan pada tempat plak,
heparin ==> memfasilitasi trombolisis
Antilipemic ==> untuk klien hiperlipidemia
'Stool softener ==> mempertahankan fungsi
bowel normal & mengurangi straining
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PATOFISIOLOGIRupturnya aterosklerosis → dikeluarkannya substansi yang dapat menstimulasi agregasi platelet,pembentukan trombus dan tonus vasokonstriktor lokal→ pembentukan trombus→ sumbatan→ aliranterhambat ke otot jantung→ iskemia→ bila lebih 20'-45‘ hipoksia irreversible→ kematian sel (infark) dannekrosis jaringan→ jaringan otot jantung mengeras→ keluarnya enzim intra sel.
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Jaringan yang nekrotik→ tdk berfungsi secara
metabolik, tidak menghasilkan dan menghantarkanlistrik juga tidak berperan dalam kontraksi mekanik→ menurunnya kontraktilitas otot jantung→ menurunnya stroke volume, CO, TD dan perfusi jaringan.
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DECOMP CORDIS/ CHF
(CHRONIC HEART FAILURE)
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PENGERTIAN Keadaan dimana jantung tidak mampu lagi
memompakan darah secukupnya dalam memenuhikebutuhan sirkulasi badan untuk keperluanmetabolisme jaringan tubuh pada keadaan tertentu,sedangkan tekanan pengisian ke dalam jantung masihcukup tinggi.
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ETIOLOGI Penyakit arteri koroner
Infark myokardium.
Kardiomyopathy primer Hipertensi
Penyakit katup jantung
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MANIFESTASI KLINIS Manifestasi gagal jantung ventrikel kiri
Dyspnea
Batuk
Fatigue, weaknes.
Nocturia, oliguria.
Bingung hilang memory.
Tahikardi
Ronchi Bunyi jantung S1 – S4
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PENATALAKSANAAN MEDIS Menurunkan preload : diuretik, dilatasi vena
(morphin, nitrogliserin) Meningkatkan preload : Pemberian cairan.
Menurunkan afterload : Vasodilator(nitropusside, nitrogliserin) Meningkatkan afterload : vasopressor
(norepinefrin) Meningkatkan kontraksi : obat simpatik
(dobutamin, dopamin), Phosphodiesteraseinhibitor (Amrinone, digitalis) Menurunkan kontraksi : beta bloker. Menstabilkan irama jantung.
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ETIOLOGI Demam reumatik akut terjadi sesudah / sebagai akibat
dari infeksi streptococcus beta hemolyticus grup Aseperti tonsilitis, faringitis, atau otitis media
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MANIFESTASI KLINIS Demam remiten yang tidak melebihi 39 derajat celcius
atau atritis yang timbul setelah 2-3 minggu setelahinfeksi
Malaise (perasaan sakit dan kurang enak badan)
Atralgia (nyeri sendi)
Pankarditis
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PENATALAKSANAAN MEDIS Eradikasi terhadap kuman streptococcus dengan
pemberian penisilin benzatin / eritromisin
Obat antiinflamasi seperti salisilat, kortikosteroiddiberikan pada pasien dengan karditis dankardiomegali seperti prednison
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PATOFISIOLOGI Terjadinya infeksi primer seperti infeksi steptococus
pada faringitis→ serangan karditis rematik akut berulang
→ kerusakan pada katup jantung→ sembuh dengan
sendiri/ kecacatan secara lambat pada katup→ penuruna
kerja jantung sampai gagal jantung
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PENGERTIAN Suatu proses inflamasi (infeksi) yang terjadi pada
bagian lapisan otot jantung yaitu bagian miokardium
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ETIOLOGI Proses infeksi oleh virus , bakteri, jarum, parasit,
protozoa, dan spiroseta atau dapat disebabkan olehkeadaan hipersensitifitas seperti demam rematik.
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MANIFESTASI KLINISGejala tergantung dari jenis infeksi, derajat
kerusakan jantung
Kelelahan Dispneu
Berdebar-debar
Suara jantung tambahan, irama galop
Demam dan tahikardi
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PENGERTIAN Peningkatan tekanan darah melebihi nilai normal
yang ditetapkan berdasarkan standar yang tlehaditentukan
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……..
4. Bidang ginjal:
Azotemia, proteinuria, oligouria.
5. Bidang obstetri
Preklampsia dg gejala berupa gangguan
penglihatan, sakit kepala hebat, kejang, nyeri
abdomen kuadran atas, gagal jantung kongestifdan oliguri, serta gangguan kesadaran/ gangguan
serebrovaskuler.
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PATOFISIOLOGI
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TROMBOPLEBITIS
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PENGERTIAN Tromboflebitis adalah invasi/perluasan
mikroorganisme patogen yang mengikuti aliran darahdisepanjang vena dan cabang-cabangnya.
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MANIFESTASI KLINIS
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PATOFISIOLOGI
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ANEMIA
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PENGERTIAN
Anemia adalah kekurangan hemoglobin (Hb). Hbadalah protein dalam sel darah merah, yangmengantar oksigen dari paru ke bagian tubuh yang
lain. Anemia didefinisikan oleh tingkat Hb. Sebagian besar
dokter sepakat bahwa tingkat Hb di bawah 6,5menunjukkan anemia yang gawat. Tingkat Hb yangnormal adalah sedikitnya 12 untuk perempuan dan 14untuk laki-laki.
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MANIFESTASI KLINIS
Anemia menyebabkan kelelahan, sesak napas dankepusingan. Orang dengan anemia merasa badannyakurang enak dibandingkan orang dengan tingkat Hb
yang wajar. Mereka merasa lebih sulit untuk bekerja. Artinya mutu hidupnya lebih rendah.
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PENATALAKSANAAN MEDIS
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PATOFISIOLOGI
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PEMBERIAN CAIRAN PARENTERAL
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PENGERTIAN
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ETIOLOGI
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PATOFISIOLOGI