ecg step y step 2015 hashem
TRANSCRIPT
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ECG STEP by STEP
Dr.Hashem Yaseen MBBS.King Abdullah University Hospital
Jordan university of science and technology
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation
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What the ECG is about?
• Electrocardiogram• It reflects the electricity of
the heart• The Heart contracts
electrical changes depolarization can be detected by skin electrodes
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Normal Impulse ConductionSinoatrial node
AV node
Bundle of His
Bundle Branches
Purkinje fibers
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The “PQRST”
• P wave - Atrial depolarization
• T wave - Ventricular repolarization (relaxation)
• QRS - Ventricular depolarization
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•Q=the first downward deflection
•R=the upward deflection
•S=any deflection below baseline following R
QRS complex
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The PR Interval
Atrial depolarization +
delay in AV junction (AV node/Bundle of His)
(delay allows time for the atria to contract before the ventricles contract)
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The ECG Paper
• Horizontally– One small box - 0.04 s– One large box - 0.20 s
• Vertically– One large box - 0.5 mV
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation• Reading 12-Lead ECG’s
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Rhythm Analysis
• Step 1: Calculate rate.• Step 2: Determine regularity.• Step 3: Assess the P waves.• Step 4: Determine PR interval.• Step 5: Determine QRS duration.
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Step 1: Calculate Rate
– Find R wave that lands on a bold line.– Count the # of large boxes to the next R wave. If
the second R wave is 1 large box away the rate is 300, 2 boxes - 150, 3 boxes - 100, 4 boxes - 75, etc. (300/# of boxes from R to R)
R wave
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Step 1: Calculate Rate
– Memorize the sequence:300 - 150 - 100 - 75 - 60 - 50
Interpretation?
300
150
100
75
60
50
Approx. 1 box less than 100 = 95 bpm
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Step 2: Determine regularity
• Look at the R-R distances (using a caliper or markings on a pen or paper).
• Regular (are they equidistant apart)? Occasionally irregular? Regularly irregular? Irregularly irregular?
Interpretation? Regular
R R
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Step 3: Assess the P waves
• Are there P waves?• Do the P waves all look alike?• Do the P waves occur at a regular rate?• Is there one P wave before each QRS?Interpretation? Normal P waves with 1 P
wave for every QRS
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Step 4: Determine PR interval
• Normal: 0.12 - 0.20 seconds. (3 - 5 boxes)
Interpretation? 0.12 seconds
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Step 5: QRS duration
• Normal: 0.04 - 0.12 seconds. (1 - 3 boxes)
Interpretation? 0.08 seconds
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Rhythm Summary
• Rate 90-95 bpm• Regularity regular• P waves normal• PR interval 0.12 s• QRS duration 0.08 sInterpretation? Normal Sinus Rhythm
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation• Reading 12-Lead ECG’s
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Normal Sinus Rhythm (NSR)
• Etiology: the electrical impulse is formed in the SA node and conducted normally.
• This is the normal rhythm of the heart; other rhythms that do not conduct via the typical pathway are called arrhythmias.
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NSR Parameters
• Rate 60 - 100 bpm• Regularity regular• P waves normal• PR interval 0.12 - 0.20 s• QRS duration 0.04 - 0.12 sAny deviation from above is sinus tachycardia,
sinus bradycardia or an arrhythmia
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Arrhythmia Formation
Arrhythmias can arise from problems in the:• Sinus node• Atrial cells• AV junction• Ventricular cells
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SA Node Problems
The SA Node can:• fire too slow• fire too fast
Sinus BradycardiaSinus Tachycardia
Sinus Tachycardia may be an appropriate response to stress.
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Atrial Cell Problems
Atrial cells can:• fire occasionally
from a focus
• fire continuously due to a looping re-entrant circuit
Premature Atrial Contractions (PACs)
Atrial Flutter
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Teaching Moment
• A re-entrant pathway occurs when an impulse loops and results in self-perpetuating impulse formation.
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Atrial Cell Problems
Atrial cells can also:• fire continuously from multiple foci orfire continuously due to multiple micro re-entrant “wavelets”
Atrial Fibrillation
Atrial Fibrillation
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AV Junctional Problems
The AV junction can:• fire continuously
due to a looping re-entrant circuit
• block impulses coming from the SA Node
Paroxysmal Supraventricular Tachycardia
AV Junctional Blocks
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Ventricular Cell Problems
Ventricular cells can:• fire occasionally from
1 or more foci• fire continuously
from multiple foci• fire continuously due
to a looping re-entrant circuit
Premature Ventricular Contractions (PVCs)
Ventricular Fibrillation
Ventricular Tachycardia
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation• Reading 12-Lead ECG’s
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Arrhythmias
• Sinus Rhythms• Premature Beats• Supraventricular Arrhythmias• Ventricular Arrhythmias• AV Junctional Blocks
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Sinus Rhythms
• Sinus Bradycardia
• Sinus Tachycardia
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Rhythm #1
30 bpm• Rate?• Regularity? regular
normal
0.10 s
• P waves?• PR interval? 0.12 s• QRS duration?
Interpretation? Sinus Bradycardia
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Sinus Bradycardia
• Deviation from NSR- Rate < 60 bpm
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Sinus Bradycardia
• Etiology: SA node is depolarizing slower than normal, impulse is conducted normally (i.e. normal PR and QRS interval).
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Rhythm #2
130 bpm• Rate?• Regularity? regular
normal
0.08 s
• P waves?• PR interval? 0.16 s• QRS duration?
Interpretation? Sinus Tachycardia
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Sinus Tachycardia
• Deviation from NSR- Rate > 100 bpm
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Sinus Tachycardia
• Etiology: SA node is depolarizing faster than normal, impulse is conducted normally.
• Remember: sinus tachycardia is a response to physical or psychological stress, not a primary arrhythmia.
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Premature Beats
• Premature Atrial Contractions (PACs)
• Premature Ventricular Contractions (PVCs)
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Rhythm #3
70 bpm• Rate?• Regularity? occasionally irreg.
2/7 different contour
0.08 s
• P waves?• PR interval? 0.14 s (except 2/7)• QRS duration?
Interpretation? NSR with Premature Atrial Contractions
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Premature Atrial Contractions
• Deviation from NSR– These ectopic beats originate in the atria
(but not in the SA node), therefore the contour of the P wave, the PR interval, and the timing are different than a normally generated pulse from the SA node.
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Premature Atrial Contractions
• Etiology: Excitation of an atrial cell forms an impulse that is then conducted normally through the AV node and ventricles.
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Teaching Moment
• When an impulse originates anywhere in the atria (SA node, atrial cells, AV node, Bundle of His) and then is conducted normally through the ventricles, the QRS will be narrow (0.04 - 0.12 s).
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Rhythm #4
60 bpm• Rate?• Regularity? occasionally irreg.
none for 7th QRS
0.08 s (7th wide)
• P waves?• PR interval? 0.14 s• QRS duration?
Interpretation? Sinus Rhythm with 1 PVC
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PVCs
• Deviation from NSR– Ectopic beats originate in the ventricles resulting
in wide and bizarre QRS complexes.– When there are more than 1 premature beats
and look alike, they are called “uniform”. When they look different, they are called “multiform”.
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PVCs
• Etiology: One or more ventricular cells are depolarizing and the impulses are abnormally conducting through the ventricles.
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Teaching Moment
• When an impulse originates in a ventricle, conduction through the ventricles will be inefficient and the QRS will be wide and bizarre.
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Ventricular Conduction
NormalSignal moves rapidly through the ventricles
AbnormalSignal moves slowly through the ventricles
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Arrhythmias
• Sinus Rhythms• Premature Beats• Supraventricular Arrhythmias• Ventricular Arrhythmias• AV Junctional Blocks
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Supraventricular Arrhythmias
• Atrial Fibrillation
• Atrial Flutter
• Paroxysmal Supraventricular Tachycardia
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Rhythm #5
100 bpm• Rate?• Regularity? irregularly irregular
none
0.06 s
• P waves?• PR interval? none• QRS duration?
Interpretation? Atrial Fibrillation
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Atrial Fibrillation
• Deviation from NSR– No organized atrial depolarization, so no
normal P waves (impulses are not originating from the sinus node).
– Atrial activity is chaotic (resulting in an irregularly irregular rate).
– Common, affects 2-4%, up to 5-10% if > 80 years old
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Atrial Fibrillation
• Etiology: Recent theories suggest that it is due to multiple re-entrant wavelets conducted between the R & L atria. Either way, impulses are formed in a totally unpredictable fashion. The AV node allows some of the impulses to pass through at variable intervals (so rhythm is irregularly irregular).
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Rhythm #6
70 bpm• Rate?• Regularity? regular
flutter waves
0.06 s
• P waves?• PR interval? none• QRS duration?
Interpretation? Atrial Flutter
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Atrial Flutter
• Deviation from NSR– No P waves. Instead flutter waves (note
“sawtooth” pattern) are formed at a rate of 250 - 350 bpm.
– Only some impulses conduct through the AV node (usually every other impulse).
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Atrial Flutter
• Etiology: Reentrant pathway in the right atrium with every 2nd, 3rd or 4th impulse generating a QRS (others are blocked in the AV node as the node repolarizes).
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Rhythm #7
74 148 bpm• Rate?• Regularity? Regular regular
Normal none
0.08 s
• P waves?• PR interval? 0.16 s none• QRS duration?
Interpretation? Paroxysmal Supraventricular Tachycardia (PSVT)
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PSVT
• Deviation from NSR– The heart rate suddenly speeds up, often
triggered by a PAC (not seen here) and the P waves are lost.
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PSVT
• Etiology: There are several types of PSVT but all originate above the ventricles (therefore the QRS is narrow).
• Most common: abnormal conduction in the AV node (reentrant circuit looping in the AV node).
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Hungry?????
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Ventricular Arrhythmias
• Ventricular Tachycardia
• Ventricular Fibrillation
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Rhythm #8
160 bpm• Rate?• Regularity? regular
none
wide (> 0.12 sec)
• P waves?• PR interval? none• QRS duration?
Interpretation? Ventricular Tachycardia
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Ventricular Tachycardia
• Deviation from NSR– Impulse is originating in the ventricles (no
P waves, wide QRS).
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Ventricular Tachycardia
• Etiology: There is a re-entrant pathway looping in a ventricle (most common cause).
• Ventricular tachycardia can sometimes generate enough cardiac output to produce a pulse; at other times no pulse can be felt.
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Rhythm #9
none• Rate?• Regularity? irregularly irreg.
none
wide, if recognizable
• P waves?• PR interval? none• QRS duration?
Interpretation? Ventricular Fibrillation
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Ventricular Fibrillation
• Deviation from NSR– Completely abnormal.
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Ventricular Fibrillation
• Etiology: The ventricular cells are excitable and depolarizing randomly.
• Rapid drop in cardiac output and death occurs if not quickly reversed
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Arrhythmias
• Sinus Rhythms• Premature Beats• Supraventricular Arrhythmias• Ventricular Arrhythmias• AV Junctional Blocks
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AV Nodal Blocks
• 1st Degree AV Block
• 2nd Degree AV Block, Type I
• 2nd Degree AV Block, Type II
• 3rd Degree AV Block
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Rhythm #10
60 bpm• Rate?• Regularity? regular
normal
0.08 s
• P waves?• PR interval? 0.36 s• QRS duration?
Interpretation? 1st Degree AV Block
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1st Degree AV Block
• Deviation from NSR– PR Interval > 0.20 s
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1st Degree AV Block
• Etiology: Prolonged conduction delay in the AV node or Bundle of His.
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Rhythm #11
50 bpm• Rate?• Regularity? regularly irregular
nl, but 4th no QRS
0.08 s
• P waves?• PR interval? lengthens• QRS duration?
Interpretation? 2nd Degree AV Block, Type I
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2nd Degree AV Block, Type I
• Deviation from NSR– PR interval progressively lengthens, then
the impulse is completely blocked (P wave not followed by QRS).
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2nd Degree AV Block, Type I
• Etiology: Each successive atrial impulse encounters a longer and longer delay in the AV node until one impulse (usually the 3rd or 4th) fails to make it through the AV node.
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Rhythm #12
40 bpm• Rate?• Regularity? regular
nl, 2 of 3 no QRS
0.08 s
• P waves?• PR interval? 0.14 s• QRS duration?
Interpretation? 2nd Degree AV Block, Type II
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2nd Degree AV Block, Type II
• Deviation from NSR– Occasional P waves are completely blocked
(P wave not followed by QRS).
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2nd Degree AV Block, Type II
• Etiology: Conduction is all or nothing (no prolongation of PR interval); typically block occurs in the Bundle of His.
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Rhythm #13
40 bpm• Rate?• Regularity? regular
no relation to QRS
wide (> 0.12 s)
• P waves?• PR interval? none• QRS duration?
Interpretation? 3rd Degree AV Block
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3rd Degree AV Block
• Deviation from NSR– The P waves are completely blocked in the
AV junction; QRS complexes originate independently from below the junction.
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3rd Degree AV Block
• Etiology: There is complete block of conduction in the AV junction, so the atria and ventricles form impulses independently of each other. Without impulses from the atria, the ventricles own intrinsic pacemaker kicks in at around 30 - 45 beats/minute.
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Remember• When an impulse originates in a ventricle,
conduction through the ventricles will be inefficient and the QRS will be wide and bizarre.
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation
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Diagnosing a MITo diagnose a myocardial infarction you need to go beyond looking at a rhythm strip and obtain a 12-Lead ECG.
Rhythm Strip
12-Lead ECG
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The 12-Lead ECG
• The 12-Lead ECG sees the heart from 12 different views.
• Therefore, the 12-Lead ECG helps you see what is happening in different portions of the heart.
• The rhythm strip is only 1 of these 12 views.
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The 12-Leads
The 12-leads include:
–3 Limb leads (I, II, III)
–3 Augmented leads (aVR, aVL,
aVF)–6 Precordial leads
(V1- V6)
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Views of the Heart
Some leads get a good view of the:
Anterior portion of the heart
Lateral portion of the heart
Inferior portion of the heart
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ST Elevation
One way to diagnose an acute MI is to look for elevation of the ST segment.
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ST Elevation (cont)
Elevation of the ST segment (greater than 1 small box) in 2 leads is consistent with a myocardial infarction.
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Anterior View of the Heart
The anterior portion of the heart is best viewed using leads V1- V4.
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Anterior Myocardial Infarction
If you see changes in leads V1 - V4 that are consistent with a myocardial infarction, you can conclude that it is an anterior wall myocardial infarction.
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Putting it all TogetherDo you think this person is having a myocardial infarction. If so, where?
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InterpretationYes, this person is having an acute anterior wall myocardial infarction.
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Other MI Locations
Now that you know where to look for an anterior wall myocardial infarction let’s look at how you would determine if the MI involves the lateral wall or the inferior wall of the heart.
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Other MI LocationsFirst, take a look again at this picture of the heart.
Anterior portion of the heart
Lateral portion of the heart
Inferior portion of the heart
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Other MI LocationsSecond, remember that the 12-leads of the ECG look at different portions of the heart. The limb and augmented leads “see” electrical activity moving inferiorly (II, III and aVF), to the left (I, aVL) and to the right (aVR). Whereas, the precordial leads “see” electrical activity in the posterior to anterior direction.
Limb Leads Augmented Leads Precordial Leads
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Other MI Locations
Now, using these 3 diagrams let’s figure where to look for a lateral wall and inferior wall MI.
Limb Leads Augmented Leads Precordial Leads
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Anterior MI
Remember the anterior portion of the heart is best viewed using leads V1- V4.
Limb Leads Augmented Leads Precordial Leads
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Lateral MISo what leads do you think the lateral portion of the heart is best viewed?
Limb Leads Augmented Leads Precordial Leads
Leads I, aVL, and V5- V6
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Inferior MINow how about the inferior portion of the heart?
Limb Leads Augmented Leads Precordial Leads
Leads II, III and aVF
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Putting it all TogetherNow, where do you think this person is having a myocardial infarction?
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Inferior Wall MIThis is an inferior MI. Note the ST elevation in leads II, III and aVF.
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Putting it all TogetherHow about now?
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Anterolateral MIThis person’s MI involves both the anterior wall (V2-V4) and the lateral wall (V5-V6, I, and aVL)!
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation• Reading 12-Lead ECG’s
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The 12-Lead ECG
The 12-Lead ECG contains a wealth of information. In Module V you learned that ST segment elevation in two leads is suggestive of an acute myocardial infarction. In this module we will cover:
– ST Elevation and non-ST Elevation MIs– Left Ventricular Hypertrophy– Bundle Branch Blocks
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ST Elevation and non-ST Elevation MIs
• When myocardial blood supply is abruptly reduced or cut off to a region of the heart, a sequence of events occur beginning with ischemia (inadequate tissue perfusion), followed by necrosis (infarction), and eventual fibrosis (scarring) if the blood supply isn't restored in an appropriate period of time.
• The ECG changes over time with each of these events…
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ECG ChangesWays the ECG can change include:
Appearance of pathologic Q-waves
T-waves
peaked flattened inverted
ST elevation & depression
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ECG Changes & the Evolving MI
There are two distinct patterns of ECG change depending if the infarction is:
–ST Elevation (Transmural or Q-wave), or–Non-ST Elevation (Subendocardial or non-Q-wave)
Non-ST Elevation
ST Elevation
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ST Elevation Infarction
ST depression, peaked T-waves, then T-wave inversion
The ECG changes seen with a ST elevation infarction are:
Before injury Normal ECG
ST elevation & appearance of Q-waves ST segments and T-waves return to normal, but Q-waves persist
Ischemia
Infarction
Fibrosis
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ST Elevation Infarction
Here’s a diagram depicting an evolving infarction:A. Normal ECG prior to MI
B. Ischemia from coronary artery occlusion results in ST depression (not shown) and peaked T-waves
C. Infarction from ongoing ischemia results in marked ST elevation
D/E. Ongoing infarction with appearance of pathologic Q-waves and T-wave inversion
F. Fibrosis (months later) with persistent Q- waves, but normal ST segment and T- waves
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ST Elevation Infarction
Here’s an ECG of an acute inferior wall MI:
Look at the inferior leads (II, III, aVF).
Question: What ECG changes do you see?
ST elevation and Q-wavesExtra credit: What is the rhythm? Atrial fibrillation (irregularly irregular with narrow QRS)!
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Non-ST Elevation Infarction
Here’s an ECG of an inferior wall MI later in time:
Now what do you see in the inferior leads?
ST elevation, Q-waves and T-wave inversion
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Non-ST Elevation Infarction
ST depression & T-wave inversion
The ECG changes seen with a non-ST elevation infarction are:
Before injury Normal ECG
ST depression & T-wave inversion
ST returns to baseline, but T-wave inversion persists
Ischemia
Infarction
Fibrosis
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Non-ST Elevation Infarction
Here’s an ECG of an evolving non-ST elevation MI:
Note the ST depression and T-wave inversion in leads V2-V6.
Question: What area of the heart is infarcting?
Anterolateral
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Left Ventricular Hypertrophy
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Left Ventricular HypertrophyCompare these two 12-lead ECGs. What stands out as different with the second one?
Normal Left Ventricular Hypertrophy
Answer: The QRS complexes are very tall (increased voltage)
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Left Ventricular HypertrophyWhy is left ventricular hypertrophy characterized by tall QRS complexes?
LVH ECHOcardiogramIncreased QRS voltage
As the heart muscle wall thickens there is an increase in electrical forces moving through the myocardium resulting in increased QRS voltage.
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Left Ventricular Hypertrophy• Specific criteria exists to diagnose LVH using a 12-lead ECG.
– For example:• The R wave in V5 or V6 plus the S wave in V1 or V2 exceeds 35
mm.
However for now, all you need to know is that the QRS voltage increases with LVH.
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Bundle Branch Blocks
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Bundle Branch BlocksTurning our attention to bundle branch blocks…
Remember normal impulse conduction is
SA node AV node Bundle of His Bundle Branches Purkinje fibers
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Normal Impulse ConductionSinoatrial node
AV node
Bundle of His
Bundle Branches
Purkinje fibers
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Bundle Branch BlocksSo, depolarization of the Bundle Branches and Purkinje fibers are seen as the QRS complex on the ECG.
Therefore, a conduction block of the Bundle Branches would be reflected as a change in the QRS complex.
Right BBB
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Bundle Branch BlocksWith Bundle Branch Blocks you will see two changes on the ECG.
1. QRS complex widens (> 0.12 sec). 2. QRS morphology changes (varies depending on ECG lead, and if it is
a right vs. left bundle branch block).
Look at the V leads to recognize Bundle Branch Blocks
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Bundle Branch BlocksWhy does the QRS complex widen?
When the conduction pathway is blocked it will take longer for the electrical signal to pass throughout the ventricles because the impulse has to travel from cell to cell inefficiently.
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Right Bundle Branch BlocksWhat QRS morphology is characteristic?
V1
For RBBB the wide QRS complex assumes a unique, virtually diagnostic shape (upright “rabbit ears”) in those leads overlying the right ventricle, V1 and V2.
“Rabbit Ears”
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Left Bundle Branch BlocksWhat QRS morphology is characteristic?For LBBB the wide QRS complex assumes a wide predominantly downward deflection leads opposite the left ventricle, i.e., V1 and V2 (right ventricular leads) and the QRS is upright and wide in V5 and V6 (may or may not be notched)
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Guess: RBBB or LBBB?
Wide QRSRabbit Ears in V1 and V2
= RBBB!
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Guess: RBBB or LBBB?
Wide QRSMostly down in V1 and V2Mostly up in lead V6
= LBBB!
Recall that in either RBBB or LBBB, the QRS must be wide (> .12 sec)
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What causes BBBs?• Coronary artery disease • Thickened, stiffened or weakened heart
muscle (cardiomyopathy) • Infection (myocarditis) of the heart muscle• Hypertension• Scar tissue after heart surgery • Congenital abnormality
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SummaryThis Module introduced you to:
– ST Elevation and Non-ST Elevation MIs – Left Ventricular Hypertrophy– Bundle Branch Blocks
Don’t worry too much right now about trying to remember all the details. You’ll focus more on advanced ECG interpretation in your clinical years!
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Learning STEPS• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation• Reading 12-Lead ECG’s
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Reading 12-Lead ECGs
• The 12-Lead ECG contains information that will assist you in making diagnostic and treatment decisions in your clinical practice. In previous modules you learned how to read and interpret parts of the ECG. Now, we will bring all that you have learned together so that you can systematically read and interpret a 12-lead ECG.
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Reading 12-Lead ECGs
The best way to read 12-lead ECGs is to develop a step-by-step approach (just as we did for analyzing a rhythm strip). In these modules we present a 6-step approach:
1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS5. Assess for HYPERTROPHY6. Look for evidence of INFARCTION
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Rate Rhythm Axis Intervals Hypertrophy Infarct
• In step 2 you learned how to calculate the rate. If you need a refresher return to that step.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
• In step 2 you learned how to systematically analyze a rhythm by looking at the rate, regularity, P waves, PR interval and QRS complexes.
• In steps 3, 4, 5 you learned how to recognize Normal Sinus Rhythm and the 13 most common rhythm disturbances.
• If you need a refresher return to these steps.
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Tip: the rhythm strip portion of the 12-lead ECG is a good place to look at when trying to determine the rhythm because the 12 leads only capture a few beats.
Lead II
Rhythm?
Atrial fibrillation
Rhythm strip
1 of 12 leads
Rate Rhythm Axis Intervals Hypertrophy Infarct
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Axis refers to the mean QRS axis (or vector) during ventricular depolarization. As you recall when the ventricles depolarize (in a normal heart) the direction of current flows leftward and downward because most of the ventricular mass is in the left ventricle. We like to know the QRS axis because an abnormal axis can suggest disease such as pulmonary hypertension from a pulmonary embolism.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
The QRS axis is determined by overlying a circle, in the frontal plane. By convention, the degrees of the circle are as shown.
The normal QRS axis lies between -30o and +90o.
0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
A QRS axis that falls between -30o and -90o is abnormal and called left axis deviation.
A QRS axis that falls between +90o and +150o is abnormal and called right axis deviation.
A QRS axis that falls between +150o and -90o is abnormal and called superior right axis deviation.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
We can quickly determine whether the QRS axis is normal by looking at leads I and II .
If the QRS complex is overall positive (R > Q+S) in leads I and II, the QRS axis is normal.
QRS negative (R < Q+S)
In this ECG what leads have QRS complexes that are negative? equivocal?
QRS equivocal (R = Q+S)
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Rate Rhythm Axis Intervals Hypertrophy Infarct
How do we know the axis is normal when the QRS complexes are positive in leads I and II ?
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Rate Rhythm Axis Intervals Hypertrophy Infarct
The answer lies in the fact that each frontal lead corresponds to a location on the circle.
0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
I
IIavF
avLavR
Limb leads
I = +0o
II = +60o
III = +120o
Augmented leads
avL = -30o
avF = +90o
avR = -150o
I
IIIII
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0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
Rate Rhythm Axis Intervals Hypertrophy Infarct
Since lead I is orientated at 0o a wave of depolarization directed towards it will result in a positive QRS axis. Therefore any mean QRS vector between -90o and +90o will be positive.
I
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0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
Rate Rhythm Axis Intervals Hypertrophy Infarct
Since lead I is orientated at 0o a wave of depolarization directed towards it will result in a positive QRS axis. Therefore any mean QRS vector between -90o and +90o will be positive.
Similarly, since lead II is orientated at 60o a wave of depolarization directed towards it will result in a positive QRS axis. Therefore any mean QRS vector between -30o and +150o will be positive.
I
II
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0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
Rate Rhythm Axis Intervals Hypertrophy Infarct
Now using what you just learned fill in the following table. For example, if the QRS is positive in lead I and negative in lead II what is the QRS axis? (normal, left, right or right superior axis deviation)
QRS Complexes
I
Axis I II + + + -
normalleft axis deviation
II
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0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
Rate Rhythm Axis Intervals Hypertrophy Infarct
…if the QRS is negative in lead I and positive in lead II what is the QRS axis? (normal, left, right or right superior axis deviation)
QRS Complexes
I
Axis I II + + + - - +
normalleft axis deviationright axis deviation
II
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0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
Rate Rhythm Axis Intervals Hypertrophy Infarct
…if the QRS is negative in lead I and negative in lead II what is the QRS axis? (normal, left, right or right superior axis deviation)
QRS Complexes
I
Axis I II + + + - - +
- -
normalleft axis deviationright axis deviation
right superior axis deviation
0o
30o
-30o
60o
-60o-90o
-120o
90o 120o
150o
180o
-150o
II
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Is the QRS axis normal in this ECG? No, there is left axis deviation.
The QRS is positive in I
and negative in II.
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Rate Rhythm Axis Intervals Hypertrophy InfarctTo summarize:
–The normal QRS axis falls between -30o and +90o because ventricular depolarization is leftward and downward.
–Left axis deviation occurs when the axis falls between -30o and -90o.–Right axis deviation occurs when the axis falls between +90o and +150o.
–Right superior axis deviation occurs when the axis falls between between +150o and -90o.
QRS ComplexesAxis I II
+ + + - - +
- -
normalleft axis deviationright axis deviation
right superior axis deviation
– A quick way to determine the QRS axis is to look at the QRS complexes in leads I and II.
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
To summarize step 7 :1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS
– Normal– Left axis deviation– Right axis deviation– Right superior axis deviation
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
NOW we will cover the next 3 steps:1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS5. Assess for HYPERTROPHY6. Look for evidence of INFARCTION
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Learning STEPS
• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation• Reading 12-Lead ECG’s
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Reading 12-Lead ECGs
In Ojective 7 we introduced a 6 step approach for analyzing a 12-lead ECG and covered the first 3 steps. In this module we will cover the last 3 steps.
1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS5. Assess for HYPERTROPHY6. Look for evidence of INFARCTION
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• Intervals refers to the length of the PR and QT intervals and the width of the QRS complexes. You should have already determined the PR and QRS during the “rhythm” step, but if not, do so in this step.
• In the following few slides we’ll review what is a normal and abnormal PR, QRS and QT interval. Also listed are a few common causes of abnormal intervals.
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PR interval
< 0.12 s 0.12-0.20 s > 0.20 s
High catecholamine states
Wolff-Parkinson-WhiteNormal AV nodal blocks
Wolff-Parkinson-White 1st Degree AV Block
Rate Rhythm Axis Intervals Hypertrophy Infarct
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QRS complex
< 0.10 s 0.10-0.12 s > 0.12 s
Normal Incomplete bundle branch block
Bundle branch blockPVC
Ventricular rhythm
Remember: If you have a BBB determine if it is a right or left BBB. If you need a refresher see Module VI.
3rd degree AV block with ventricular escape rhythm
Incomplete bundle branch block
Rate Rhythm Axis Intervals Hypertrophy Infarct
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QT interval
The duration of the QT interval is proportionate to the heart rate. The faster the heart beats, the faster the ventricles repolarize so the shorter the QT interval. Therefore what is a “normal” QT varies with the heart rate. For each heart rate you need to calculate an adjusted QT interval, called the “corrected QT” (QTc):
QTc = QT / square root of RR interval
Rate Rhythm Axis Intervals Hypertrophy Infarct
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QTc interval
< 0.44 s > 0.44 s
Normal Long QT
A prolonged QT can be very dangerous. It may predispose an individual to a type of ventricular tachycardia called Torsades de Pointes. Causes include drugs, electrolyte abnormalities, CNS disease, post-MI, and congenital heart disease.
Torsades de Pointes
Long QT
Rate Rhythm Axis Intervals Hypertrophy Infarct
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Rate Rhythm Axis Intervals Hypertrophy Infarct
PR interval? QRS width? QTc interval?0.08 seconds0.16 seconds 0.49 seconds
QT = 0.40 s
RR = 0.68 s
Square root of RR = 0.82
QTc = 0.40/0.82 = 0.49 s
Interpretation of intervals? Normal PR and QRS, long QT
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Tip: Instead of calculating the QTc, a quick way to estimate if the QT interval long is to use the following rule:
A QT > half of the RR interval is probably long.
Normal QT Long QT
QT
RR
10 boxes
23 boxes 17 boxes
13 boxes
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Rate Rhythm Axis Intervals Hypertrophy Infarct
In this step of the 12-lead ECG analysis, we use the ECG to determine if any of the 4 chambers of the heart are enlarged or hypertrophied. We want to determine if there are any of the following:
– Right atrial enlargement (RAE)– Left atrial enlargement (LAE)– Right ventricular hypertrophy (RVH)– Left ventricular hypertrophy (LVH)
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Rate Rhythm Axis Intervals Hypertrophy Infarct
• In step 4 we introduced the concept of left ventricular hypertrophy. As you remember the QRS voltage increases with LVH and is characterized by tall QRS complexes in certain leads. Similarly for right ventricular hypertrophy we look at the QRS complexes for changes in voltage patterns.
• With right and left atrial enlargement we analyze the P wave (since the P wave represents atrial depolarization). Here we also look for changes in voltage patterns.
• Note: as mentioned in step 4 criteria exists to diagnose LVH, the same goes for RAE, LAE and RVH. In the following slides we will be presenting criteria you can use.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Right atrial enlargement – Take a look at this ECG. What do you notice about the P waves?
The P waves are tall, especially in leads II, III and avF. Ouch! They would hurt to sit on!!
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Right atrial enlargement – To diagnose RAE you can use the following criteria:
• II P > 2.5 mm, or• V1 or V2 P > 1.5 mm
Remember 1 small box in height = 1 mm
A cause of RAE is RVH from pulmonary hypertension.
> 2 ½ boxes (in height)
> 1 ½ boxes (in height)
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Left atrial enlargement – Take a look at this ECG. What do you notice about the P waves?
The P waves in lead II are notched and in lead V1 they have a deep and wide negative component.
Notched
Negative deflection
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Left atrial enlargement – To diagnose LAE you can use the following criteria:
• II > 0.04 s (1 box) between notched peaks, or• V1 Neg. deflection > 1 box wide x 1 box deep
Normal LAE
A common cause of LAE is LVH from hypertension.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Right ventricular hypertrophy– Take a look at this ECG. What do you notice about the axis and QRS
complexes over the right ventricle (V1, V2)?
There is right axis deviation (negative in I, positive in II) and there are tall R waves in V1, V2.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Right ventricular hypertrophy– Compare the R waves in V1, V2 from a normal ECG and one from a
person with RVH.– Notice the R wave is normally small in V1, V2 because the right
ventricle does not have a lot of muscle mass.– But in the hypertrophied right ventricle the R wave is tall in V1, V2.
Normal RVH
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Right ventricular hypertrophy – To diagnose RVH you can use the following criteria:
• Right axis deviation, and• V1 R wave > 7mm tall
A common cause of RVH is left heart failure.
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Left ventricular hypertrophy– Take a look at this ECG. What do you notice about the axis and QRS
complexes over the left ventricle (V5, V6) and right ventricle (V1, V2)?
There is left axis deviation (positive in I, negative in II) and there are tall R waves in V5, V6 and deep S waves in V1, V2.
The deep S waves seen in the leads over the right ventricle are created because the heart is depolarizing left, superior and posterior (away from leads V1, V2).
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Left ventricular hypertrophy– To diagnose LVH you can use the following criteria*:
• R in V5 (or V6) + S in V1 (or V2) > 35 mm, or• avL R > 13 mm
A common cause of LVH is hypertension.
* There are several other criteria for the diagnosis of LVH.
S = 13 mm
R = 25 mm
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Rate Rhythm Axis Intervals Hypertrophy Infarct
A 63 yo man has longstanding, uncontrolled hypertension. Is there evidence of heart disease from his hypertension? (Hint: There a 3 abnormalities.)
Yes, there is left axis deviation (positive in I, negative in II), left atrial enlargement (> 1 x 1 boxes in V1) and LVH (R in V5 = 27 + S in V2 = 10 > 35 mm).
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Rate Rhythm Axis Intervals Hypertrophy Infarct
• When analyzing a 12-lead ECG for evidence of an infarction you want to look for the following:
– Abnormal Q waves– ST elevation or depression– Peaked, flat or inverted T waves
• These topics were covered in Modules V and VI where you learned:
– ST elevation (or depression) of 1 mm in 2 or more contiguous leads is consistent with an AMI
– There are ST elevation (Q-wave) and non-ST elevation (non-Q wave) MIs
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Rate Rhythm Axis Intervals Hypertrophy Infarct
Tip: One way to determine if Q waves (and R waves) are abnormal is by looking at the width and using the following mantra (read red downwards): Any Any Q wave in V1
Any Any Q wave in V2Any Any Q wave in V3
20 A Q wave > 20 msec in V4 (i.e. 0.02 sec or ½ width of a box)30 A Q wave > 30 msec in V530 A Q wave > 30 msec in V6
30 A Q wave > 30 msec in I30 A Q wave > 30 msec in avL30 A Q wave > 30 msec in II30 A Q wave > 30 msec in avF
R40 A R wave > 40 msec in V1R50 A R wave > 50 msec in V2
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Rate Rhythm Axis Intervals Hypertrophy Infarct
This mantra corresponds to the ECG in the following way:
Any
Any
Any
20
30
30
30
3030
30
R40
R50
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
To summarize:1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS
– Normal– Left axis deviation– Right axis deviation– Right superior axis deviation
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
To summarize:1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS
– PR– QRS– QT
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
To summarize:1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS5. Assess for HYPERTROPHY
– Right and left atrial enlargement– Right and left ventricular hypertrophy
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
To summarize:1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS5. Assess for HYPERTROPHY6. Look for evidence of INFARCTION
– Abnormal Q waves– ST elevation or depression– Peaked, flat or inverted T waves
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
To summarize:1. Calculate RATE2. Determine RHYTHM3. Determine QRS AXIS4. Calculate INTERVALS5. Assess for HYPERTROPHY6. Look for evidence of INFARCTION
Now to finish this part lets analyze a 12-lead ECG!
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
A 16 yo young man ran into a guardrail while riding a motorcycle. In the ED he is comatose and dyspneic. This is his ECG.
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
What is the rate? Approx. 132 bpm (22 R waves x 6)
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
What is the rhythm? Sinus tachycardia
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
What is the QRS axis? Right axis deviation (- in I, + in II)
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
What are the PR, QRS and QT intervals?
PR = 0.12 s, QRS = 0.08 s, QTc = 0.482 s
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
Is there evidence of atrial enlargement?
No (no peaked, notched or negatively deflected P waves)
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
Is there evidence of ventricular hypertrophy?
No (no tall R waves in V1/V2 or V5/V6)
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
Infarct: Are there abnormal Q waves?
Yes! In leads V1-V6 and I, avL
Any
Any
Any
20
30
30
30
3030
30
R40
R50
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
Infarct: Is the ST elevation or depression?
Yes! Elevation in V2-V6, I and avL. Depression in II, III and avF.
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
Infarct: Are there T wave changes?
No
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SUMMARY Rate Rhythm Axis Intervals Hypertrophy Infarct
ECG analysis: Sinus tachycardia at 132 bpm, right axis deviation, long QT, and evidence of ST elevation infarction in the anterolateral leads (V1-V6, I, avL) with reciprocal changes (the ST depression) in the inferior leads (II, III, avF).
This young man suffered an acute myocardial infarction after blunt trauma. An echocardiogram showed anteroseptal akinesia in the left ventricle with severely depressed LV function (EF=28%). An angiogram showed total occlusion in the proximal LAD with collaterals from the RCA and LCX.
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Other Abnormalities• Electrolytes disturbanceso potassiumo Magnesium o Sodiumo Calcium• Pulmonary embolism • Digoxin• Pericarditis
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Electrolytes Abnormalities
• low potassium : flattened T wave and appearance of U wave at the end of T wave
• High potassium : peaked T wave with disappearance of ST segment and widened QRS complex
• same as Magnesium • Sodium has no effect on ECG• low Calcium : prolonged QT interval• high Calcium : shortened QT interval
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Elec. AbnormalitiesHYPERKALEMIA
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Elec. AbnormalitiesHYPOKALEMIA
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Elec. Abnormalities
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Elec. AbnormalitiesHYPERCALCEMIA/HYPOCALCEMIA
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Pulmonary Embolism
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Digoxin
• Digoxin : T wave
inversion with sloping depression of the ST segment..
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Digoxin
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Pericarditis
• is an inflammation of the pericardium• four stages can be distinguished on the ECG:• stage I : ST elevation in all leads. PTa depression (depression between the end of the P-wave and the beginning of the QRS- complex)• stage II : pseudonormalisation (transition)• stage III : inverted T-waves• stage IV : normalisation
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Pericarditis
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Pericarditis
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How to record an ECG???
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GOOD LUCK
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ECG STEP by STEP
Thanks Dr.Hashem Yaseen MBBS.
King Abdullah University HospitalJordan university of science and technology