NOTES

1 St. Pierre, R. An evaluation of the Nutrition Education and Training Program: Project summary. Cambridge, Mass.: Abt Associates Inc., 1981.

2 Ferb, T., 1. Glotzer, J. Nestor, and D. Napior. The Nutrition Education and Train­ing Program: A status report; 1977-1980. Cambridge, Mass.: Abt Associates Inc., 1980.

3 S1. Pierre, R., J. Glotzer, T. Cook, and R. Straw. An evaluation of the Nutrition Educa­tion and -Training Program: Nebraska's Ex­perience Nutrition Curriculum. Cambridge, Mass.: Abt Associates Inc., 1981.

4 S1. Pierrre, R., and J. Glotzer. An evaluation of the Georgia Nutrition Education and Training Program. Cambridge, Mass.: Abt Associates Inc., 1981.

5 Majure, W. Evaluation report: Eight-state consortium. Red Oak, Iowa: Experience Education, Spring 1980.

6 Swanson Center for Nutrition. Nutrition

An exceptionally readable and infor­mative review article by S.L. Taylor (Food Technology 36[3]:65-68 passim, 1982) of­fers valuable background material for inter­preting claims and counterclaims about car­cinogens in foods. Taylor discusses the mechanisms of carcinogenesis and muta-

N. N. Abumrad et al. (American Journal of Clinical Nutrition 34:2551-59, 1981) describe a patient on long-term parenteral nutrition who apparently suffered from molybdenum deficiency. The clinical symp­tomology, which included nausea and pro­gressed to edema and coma, was more se­vere when the infusion mixture contained pure amino acid solutions than when it con­tained protein hydrolysates or plasma. Bio­chemical symptoms, which worsened with infusion of methionine or bisulfite, in-

66 JOURNAL OF NUTRITION EDUCATION

education field test evaluation report. Omaha, Nebr. : Swanson Center for Nutri­tion, July 1979.

7 Emory University. Georgia educational model for nutrition education and manage­ment training: Final report. Atlanta, Ga.: Program in Dietetics, Division of Allied Health Professions, Emory University School of Medicine, July 1980.

8 Wolff, H. Evaluation of the curriculum guide for nutrition education: Preschool through grade six. Report prepared for the California State Department of Education, Nutrition Education and Training Program, September 29,1980.

9 West Virginia Department of Education, Bureau of Federal Programs and Services, Bureau of Planning, Research and Evalua­tion. Evaluation report: The nutrition educa­tion team: Final report. Charleston, W.V.: West Virginia Department of Education, 1977.

10 Applied Management Sciences. Evaluation

FOOD TOXICANTS

genesis and critically examines the reliability of short-term mutagenicity. He points out that the mutagenicity studies yield some false positive and some false negative results, with the degree of reliability in­fluenced by the class of chemical com­pounds as well as several aspects of experi-

MOLYBDENUM

dicated abnormalities in the metabolic pathways involved in catabolism of sulfur amino acids and purines. Since two en­zymes in these pathways- sulfite oxidase and xanthine oxidase - are molybdenum­containing proteins, the investigators ad­ministered intravenous molybdenum, a trace element that is not added intentionally to total parenteral nutrition formulas but which may be present in trace amounts in protein hydrolysates or plasma. Plasma and urinary uric acid promptly increased.

of a comprehensive nutrition education cur­riculum. Silver Spring, Md. : Applied Management Sciences, 1976.

LITERATURE CITED

1 Maretzki , A.M. A perspective on nutrition education and training. Journal of Nutrition Education 11:176-80, 1979.

2 Nestor, J . and J. Glotzer, eds. Teaching nutrition. Cambridge, Mass.: Abt Books, 1981.

3 Gillespie, A. A theoretical framework for studying school nutrition education pro­grams. Journal of Nutrition Education 13:150-53, 1981.

4 Shannon, B., P. Bell, E. Marbach, L. Hsu O'Connell, K. Graves, and R. Nicely, Jr. A K-6 nutrition curriculum evaluation­Instruction and teacher preparation. Journal of Nutrition Education, 13:9-13, 1981.

mental protocol. Despite the usefulness of mutagenicity tests for rapid screening of chemicals for potential carcinogenicity, Taylor cautions that such tests do not offer absolute proof of toxicity or of safety.

S.M.O.

Urinary sulfite excretion decreased and sulfate excretion increased. The clinical symptoms improved as well. This case sup­ports the nutritional essentiality of molyb­denum in a human subject. Also, similar to previous cases of zinc, copper, or chro­mium deficiency, it points out the impor­tance of monitoring trace element content of formulas used for long-term total parenteral nutrition. S.M. O.

VOLUME 14 NUMBER 2 1982