unidade acadêmica de medicina veterinária · 2017-06-13 · slide 1 ectima contagioso...
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Slide 1
Universidade Federal de Campina GrandeCentro de Saúde e Tecnologia Rural
Unidade Acadêmica de Medicina Veterinária
ECTIMA CONTAGIOSO
Prof. Dr. Silvano Higino
13/06/2017 ECTIMA CONTAGIOSO 1
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Slide 2
Família Poxviridae
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Slide 3
EtiologiaDoença viral altamente contagiosa de ovinos ecaprinos caracterizada pelo desenvolvimento delesões crostosas na pele do focinho e lábios e,menos frequentemente, em outros locais;
Parapoxvirus;
Boqueira, dermatite labial contagiosa;
Zoonose ocupacional.
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Fonte: www.farmpoint.com.br
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Slide 4
Etiologia
Sensibilidade: Luz, formalina, fenol, ácidos, calor 60º por30min;
Resistência - sobrevive em crostas por 11 meses, aprisco pormais de 1 ano após a retirada de animais doentes;
Estritamente dérmica.
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Slide 5
Epidemiologia
A doença afeta primariamente ovinos e caprinos;
Casos esporádicos foram relatados em bovinos e caninos;
As lesões são dolorosas, mas são benignas e somente emraras ocasiões ocorre uma reação sistêmica grave.
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Slide 6
Epidemiologia
A morbidade pode chegar a 100%, mas a mortalidade raramente
excede a 1%;
As mortes ocorrem, por invasão das larvas de moscas e bactérias
como Fusobacterium necrophorum, Dermatophilus congolensis e
Staphylococcus spp.;
Nesses casos, a mortalidade pode chegar a 50%.
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Slide 7
Patogenia
O vírus se multiplica com facilidade no epitélio da pele e das
mucosas da boca e do esôfago;
A pele intacta oferece uma barreira à penetração do vírus;
Para ocorrer a infecção é necessária a presença de pequenas abrasões
ou escarificações.
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Slide 8
Patogenia
A infecção ocorre somente quando ovírus penetra no estrato espinhoso daepiderme;
3 dias após a penetração háavermelhamento no local.
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Fonte: https://static.todamateria.com.br/upload/55/71/55715e021cccf-pele-humana.jpg
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Slide 9
Patogenia
Solução de continuidade
Camada espinhosa
Codificação de proteína homóloga ao fator de
crescimento endotelial vascular
Vascularização nas lesões
Corpúsculos de Guarnieri
Multiplicação da epiderme
Aumento na queratina
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Slide 10
Patogenia
Lesões proliferativas da epiderme iniciam por aumento dasmitoses na camada germinativa;
Em três dias o epitélio triplica sua espessura normal. Nãohavendo complicações, a lesão regride completamente emcerca de 25 dias.
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Slide 11
Patogenia
Máculas e eritema;
Pápulas;
Transformação em pústulas;
Úlceras (3 a 7 dias);
Dessecam e originam crostas;
Pode ocorrer seu desprendimento com cicatrização ou
infecção secundária.
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Slide 12
Transmissão
Contato direto, fômites, pastagenscontaminadas;
Cordeiros infectados podem contaminaros tetos das ovelhas e, dessa maneira,disseminar o vírus a outro cordeiro damesma ovelha.
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Fonte: www.farmpoint.com.br
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Slide 13
Sinais clínicos
P.I. – 2 a 4 dias;
As primeiras lesões desenvolvem-se nanas comissuras labiais;
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Fonte: www.farmpoint.com.br
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Slide 14
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Fonte: www.farmpoint.com.brFonte: www.farmpoint.com.br
Fonte: www.farmpoint.com.br
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Fonte: www.farmpoint.com.brFonte: www.farmpoint.com.br
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Slide 16
Sinais clínicos
Daí, as lesões disseminam-se para o focinho, fossas nasais eao redor dos olhos;
Em casos mais graves, as lesões ocorrem nas gengivas,almofadinha dental, palato, língua, esôfago, virilha, axila,vulva, ânus, prepúcio e membros;
Convalescentes - sólida imunidade por 2 a 3 anos;
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Fonte: Mertens, L. 2013
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Slide 18
DiagnósticoSinais clínicos e epidemiologia;
A confirmação laboratorial não é usualmente necessária;
Observação das partículas virais (crostas) e corpúsculos de inclusão pelamicroscopia eletrônica (fibroblastos de embrião de galinha);
PCR.
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Slide 19
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Fonte: en.wikipedia.org
Fonte: en.wikipedia.org Fonte: en.wikipedia.org
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Slide 20
Tratamento
Não há tratamento específico;
As lesões podem ser tratadas com uso tópico de soluções de sulfato
de cobre a 5% e iodo a 7%;
As infecções secundárias podem ser tratadas com antibióticos;
Quando os surtos ocorrem em épocas de miíases, é recomendável o
uso de repelentes.
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Slide 21
Controle e profilaxia
A profilaxia é realizada através de vacinação anual;
A vacina é produzida com vírus vivo e seu uso introduz ovírus na propriedade;
Por isso a vacinação não deve ser usada em rebanhos ondenunca ocorreu a doença;
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Slide 22
Via de Administração: Cutânea por escarificação da
pele;
Posologia: 1 gota da vacina reidratada;
Cordeiros: 1ª vacinação até 8 semanas de idade
Em zonas de alto risco: revacinar a cada 5 ou 6 meses;
Em zonas de baixo risco: única vacinação é suficiente;
Obs: não vacinar fêmeas 7 semanas antes do parto e não
vacinar ovelhas com cordeiros ao pé.
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Fonte: http://country.cdn.cevaws.com/var/ezflow_site/storage/images/media/ceva-brazil/images/ectimavac/1039761-1-por-BR/ectimavac_medium.png
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Controle e profilaxia
A imunidade ocorre em 3 semanas após a vacinação e durapor 6-8 meses;
A vacina deve ser aplicada na axila para evitar que ainfecção se dissemine por lambedura;
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Slide 24
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Epidemiologia e Dados recentes
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Slide 25
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Slide 27
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Slide 28
Obrigado!!!
www.profhigino.webnode.com
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