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Cardiovascular System
Chapter 8
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Cardiovascular System
Functions:
pump saturated oxygenated blood into
arterial system cells
pump desaturated deoxygenated blood to
lungs via veins for reoxygenation
Heart size depends on persons size
size of fist
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Anatomy of the Heart
Four Chambers: Right atrium
Left atrium
Right ventricle
Left ventricle
Four Valves:
Two atrioventricular (AV)
1. tricuspid
2. mitral
Two semilunar (SL)
1. pulmonic
2. aortic
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Heart
Chambers
Right
Atrium
Left
Atrium
Right
Ventricle
Left
Ventricle
Apex
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2 Types of Valves
AV valves - mitral between LA & LV
- tricuspid between RA & RV
Prevents backflow during contraction -systole
Semilunar valves - prevent backflow during relaxation of ventricles - diastole
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Heart Valves
Apex
Left
Right
Tricuspid
Mitral
Pulmonary Veins
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The right ventricle occupies most of the anterior surface of the cardiac surface.
Inferior border lies below the junction of the sternum and the xiphoid process.
RV narrowed superiorly and meets at 3ed costal cartilage.
LV : apical Impulse 5th ICS MCL.
RA and LA not identified in the physical examination.
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Coronary Circulation
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Four Chambers
Right atrium (deoxygenated bld)
Right ventricle (to lungs)
Left atrium (receives oxygenated blood)
Left ventricle (to systemic circulation)
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Pulmonary and Systemic Circuits
Systemic Circuit
Left side of heart
Pumps oxygenated
blood to body via
arteries
Returns deoxygenated
blood to right heart via
veins
Pulmonary Circuit
Right side of heart
Pumps deoxygenated
blood to lungs via
pulmonary arteries
Returns oxygenated
blood to left heart via
pulmonary veins
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Cardiac Cycle
It has two phases:
(A) Diastole – ventricles relax & fill with
blood (This is 2/3 of the
cardiac cycle.)
(B) Systolic – heart contracts & pushes
blood out of the ventricles to:(i) the lungs
(ii) systemic arteries
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Cardiac Cycle
Systole & Diastole = 1 heartbeat
Systole - period when ventricles
contract & eject blood
- mitral & tricuspid close
- S1 produced
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Cardiac Cycle
Diastole - ventricles in relaxed state
- atria contract blood to
ventricles
- aortic & pulmonary valve close
- S2 produced
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Heart Sounds
S1 – when closure of the AV valves
(tricuspid & mitral) & ventricles
contract
S2 – when closure of the semilunar
valves ( pulmonic & aortic) &
the ventricles relax
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Extra Heart SoundsS3 – This occurs immediately after S2
Why? Resistance to filling of ventricles
Note: also called a ventricular gallop
*It is caused by overload.
* use diaphragm (it is a high sound)
S4 - This occurs at the end of diastole, just before the next S1.
Why? The atrium contract & push blood into a non-compliant ventricles.
Note: also called an atrial gallop
*caused by HTN, CAD, Aortic stenosis, cardiomyopathy
* Use bell to listen as it is a low sound.
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Heart murmur Distinguished from heart sound by
their longer duration.
Indicate serious heart disease.
Cause by a stenotic valver orifice (abnormal narrowed).
A systolic murmur may occure with normal heart or with heart disease.
Diastole murmur always indicate heart disease.
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Murmurs
Caused by ―turbulence‖
Therefore we hear a gentle blowing, swooshing sound.
Why?
1. Velocity of blood increases
(eg. exercise, thyrotoxicosis)
2. Velocity of blood decreases (eg. anemia)
3. Structural defect in the valves or an unusual
opening occurs in the chambers
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Grading of MurmursUse VI point grading scale & record as a fraction
(ie. I/VI or II/VI)
Grades:
Grade I – barely audible, heard only in a quiet room & then with
difficulty
Grade II – clearly audible, but faint
Grade III – moderately loud, easy to hear
Grade IV – loud, associated with a thrill palpable on the chest wall
Grade V – very loud, heard with one corner of the stethoscope lifted
off the chest wall
Grade VI – loudest, still heard with the entire stethoscope lifted off
the chest
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1-SA Node (Pacemaker)
2-AV Node
3-Bundle of His
4-Perkinje fibers
Conduction System
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Conduction System of the Heart
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Electrocardiogram
Records electrical activity of the heart
P wave
Atrial depolarization
QRS complex
Ventricular depolarization
T wave
Ventricular repolarization
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EKG
Electrical activity recorded as specific waves.
Records 2 Electrical Events:
1. Depolarization - spread of impulse through
heart
2. Repolarization - return of heart muscle to
resting state.
Sequence of R & D = ACTION POTENTIAL
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EKG other terms:
Conductivity - cells ability to conduct & transmit
electrical impulse
Contractility - ability of fibers to contract
- contractility = C.O.
EKG
P waves
QRS complex
ST segment
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The heart as a pump
Cardiac Output: The amount of blood pumped
from the left ventricle each minute
Cardiac Output = Heart Rate X Stroke Volume
Heart Rate
Number of times ventricles contract each minute
Normal adult 60-100
Stroke Volume
Amount of blood ejected by the ventricles during
each systole
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Stroke Volume (SV) determined by
Preload
Contractility
Afterload
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Preload
Degree of myocardial fiber stretch at the end of diastole and
just before contraction
Determined by LVED pressure and blood return from the
venous system
Starlings Law: the more the heart fills during diastole, the
more forcefully it contracts
Afterload
Pressure or resistance that the ventricles must overcome to eject blood through the semilunar valves and into the peripheral blood vessels.
Contractility
The force of cardiac contraction independent of preload
Increased by:Sympathetic stimulation,Calcium release
Decreased by: Hypoxia and acidemia
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Preload and Afterload
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Blood Pressure
Measure of pressure exerted by blood
against walls of arteries.
Systolic - pressure when heart contracts
Diastolic - pressure with relaxation
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Postural BP Changes
Compare and contrast normal
and abnormal blood pressure
responses to postural position
changes.
What do orthostatic changes
indicate?
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Jugular venous pressure & pulses Systematic venous pressure is much
lower than arterial pressure.
Wall of veins contain less smooth muscle than arterial wall.
Venous pressure fall when left venticular out put or blood volume is significantly reduced.
Pressure in the jugular vein reflect right atrial pressure.
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The Neck Vessels
The Carotid Artery
The Jugular Venous Pulse & Pressures
2 components: (a) internal jugular
(b) external jugular
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Neck Vessels
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The Health Hx
Common or Concerning Symptoms
Chest pain.
Palpitation.
Shortness of breath: dyspnea, orthopnea, or paroxysmal nocturnal dyspnea.
Swelling or edema.
Assessment. History - major symptom, chest pain.
P - exercise, straining, activity, emotional
- rest, O2
Q - crushing, heavy, dull, burning, pressure
(own words)
R - (L) Anterior chest
- (R) arm, jaw, neck, shoulders
S - anorexia, N & V
- SOB, anxious, sweaty, dizzy
T - constant, intermittent, sudden, insidious
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Palpitation Palpitations are an unpleasent
awareness of the heart beat.
Patients use various terms such as: Skipping, Racing, Pounding, Fluttering, or Stopping the heart.
Are you ever aware of your heart beat?.
Ask Pt about the rhythem( was it fast or slow, regular, irregular).
Shortness Of Breath Dyspnea is uncomfortable awarness of breathing that
is inappropriate to a given level if exertion.
Orthopnea is duspnea that occurs when the Pt lying
down and improves when the Pt sits up.
Paroxysmal Nocturnal Dyspnea PND, describes
epispdes of sudden dyspnea & orthopnea that awaken
the Pt from sleep, usually 1or 2 hrs after going to bed,
prompting the Pt to sit up, stand up, or go to window
for air. There may be associated with wheezing &
coughing.
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Assessment continuo……
Cough
Fatique
Cyanosis or pallor
Edema
Past cardiac history
Family cardiac history
Personal habits
Environment
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Additional Assessment Areas Health perception and management
Nutrition and metabolism
Elimination
Activity and exercise
Sleep rest patterns
Cognition and perception
Self-perception and self-concept
Roles and relationships
Sexuality and reproduction
Coping and stress tolerance 10/13/2010 46Ra'eda Almashagba
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Physical Exam
Cyanosis
clubbing
edema
capillary refill
pulse - rate, rhythm, strength
pulse pressure - 120/80 pp - 40
General appearance - distress, color, LOC
Urinary output - kidney perfusion
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Assessing for Clubbing
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Recommended Sequencefor assessing cardiovascular system
1. Pulses & BP
2. Extremities
3. Neck Vessels
4. Precordium
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Heart Rate or Pulse - Evaluate for
Rate 60 - 100 Adults
90 - 120 Children
70 - 170 Newborns
< normal = bradycardia
> normal = tachycardia
Rhythm Regular or irregular
Irregular beat may indicate arrhythmias
Strength Bounding? Arteriosclerosis
Weak and thready? shock
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Blood Pressure Classification Category Systolic (mmHg) Diastolic
(mmHg)
HypertensionStage 3 (sever)Stage 2
(moderate)Stage 1 ( mild)
≥ 180160-179140-159
≥ 110100-10990-99
High Normal 130-139 85-89
Normal < 130 < 85
Optimal < 120 < 80
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The Neck Vessels
A. Carotid Arteries Palpate low in neck to avoid the sinus
Be gentle
Palpate only one side at a time to avoid compromising blood flow to the head
Auscultate using the bell
Listen in 3 places: angle of jaw
midcervical area
base of neck
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Assessment of the Jugular Vein
Purpose: To measure the ―central venous
pressure‖
Method: Position patient @ 45 degree angle at
the hip, Turn head slightly away, Use a strong
light tangentially, Observe the external jugular
over the sternomastoid muscle
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Specific Process for CVP Measurement
Locate the internal jugular pulsation
Mark the highest point of pulsation
Locate the ―angle of Louis‖
Make a ―T square‖ with 2 index cards
Read the level of intersection
Note: The normal jugular venous pressure is
2 cm or less above the sternal angle.
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Jugular Vein Pressure
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Elevated JVP: Right-sided CHF, constrictive pericarditis, tricuspid stenosis, or superior vena cava obstruction.
Low JVP: Hypovolemia.
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Hepatojugular Reflux
This is measured if the CVP (central venous pressure) is elevated or CHF is suspected.
Patient is supine
Instruct patient to breathe quietly with mouth open
With rt. hand on the patient’s RUQ of abdomen, just below the rib cage, exert firm consistent pressure for 30 seconds
Watch the level of the jugular pressure
Note: Normally the jugular rises but recedes back.
Abnormally, the pressure elevates & stays.
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Hepatojugular Reflex
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Congestive Heart Failure( CHF)
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The Heart ( Precordium)
Inspection: Check pulsations, heaves, lifts
(You may see the apical pulse.)
Note: The apical is located in the 4th or 5th
ICS @ the left MCL .
Palpate: Feel the apical impulse (also called the PMI). * Use 1 finger pad.
Use palmar side of 4 fingers to feel for other pulsations on the chest.(eg. ―thrills‖
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Palpation
Note any thrills
Palpate the PMI
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Palpation
PercussionPercussion: To check for heart enlargement
(Note:often done by chest Xray)
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Auscultation
1. Position the patient supine with the head of the table slightly elevated.
2. Always examine from the patient's right side. A quiet room is essential.
3. Listen with the diaphragm at the right 2nd intercostal near the sternum (aortic area).
4. Listen with the diaphragm at the left 2nd intercostal near the sternum (pulmonic area).
5. Listen with the diaphragm at the left 3rd, 4th, and 5th interspaces near the sternum (tricuspid area).
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Auscultation 6. Listen with the diaphragm at the apex (mitral area). 7. Listen with the bell at the apex. 8. Listen with the bell at the left 4th and 5th intercostal near
the sternum. 9. Have the patient roll on their left side.
Listen with the bell at the apex. This position brings out S3 and mitral murmurs.
10. Have the patient sit up, lean forward, and hold their breath in exhalation. Listen with the diaphragm at the left 3rd and 4th
intercostal near the sternum. This position brings out aortic murmurs.
11. Record S1, S2.12. Auscultate the carotid arteries.
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Heart Assessment (continued)
Auscultation: Start at the base of the heart.
―APE to Man‖
Aortic - 2nd Rt. ICS
Pulmonic – 2nd left ICS
Erb’s Point
Tricuspid – left sternal border
Mitral – 5th ICS @ left MCL
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Sequence for Auscultating
A. Begin with the diaphragm.
Note at each area:
1. rate & rhythm
2. identify S1 and S2
3. assess S1 and S2 separately
4. listen for extra heart sounds (ie. S3,S4)
5. listen for murmurs
B. Repeat above using the bell.
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What do you hear?
S1 and S2 sound like ―lub-dup‖
S1 is louder than S2 at the apex
S2 is louder than S1 at the base
S1 coincides with the carotid pulsation
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Auscultatory Areas
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Relationship of auscultatory finding to the chest wall
Sounds & murmurs arising from the mitral valve are usually heared best at & around the cardiac apex.
The sound originated in the tricuspid valve are heard best at or near the lower left sternal border.
Murmur arising from pulmonic valve heard best in the 2nd left ICS close to the sternum.
The sound originated in the aortic valve may heard any where from Rt 2nd ICS to the apex.
The base of the heart refers to the Rt & Lt 2nd
ICS close to the sternum.
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First and Second Heart sound
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Sounds – S1(Lub)…& S2(Dub)
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Extra Heart Sounds
S3…
Due to Rapid ventricularfilling: ventricular gallop
S1 -- S2-S3 (Ken--tuc-ky)
S4…
Due to slow ventricular
contraction: atrial gallop
S4-S1 — S2 (Ten-nes—
see)
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Murmurs
turbulent blood flow
within the heart
Listen for murmurs in
the same auscultatory
sites APETM
Grading of murmurs
(I/VI -VI/VI)
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THE END
Thank You
Ra'eda Almashagba